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Article

Activation of γ-globin gene expression by GATA1 and NF-Y in hereditary persistence of fetal hemoglobin

1 Aug 2021

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Share to Hemoglobinopathies and Related Disorders
Share to Prenatal Screening and Diagnostics
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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 34341563
DOI: 10.1038/s41588-021-00904-0

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Activation of γ-globin gene expression by GATA1 and NF-Y in hereditary persistence of fetal hemoglobinPMID 34341563Read ↗

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Update on fetal hemoglobin gene regulation in hemoglobinopathiesSemantic proximity Read ↗

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Regulation of human fetal hemoglobin: new players, new complexitiesSemantic proximity Read ↗

Mechanisms of Globin Gene Regulation in MammalsSemantic proximity Read ↗

Regulation of embryonic/fetal globin genes by nuclear hormone receptors: a novel perspective on hemoglobin switching.Semantic proximity Read ↗

ZNF410 represses fetal globin by singular control of CHD4Semantic proximity Read ↗

Fetal globin gene repressors as drug targets for molecular therapies to treat the β-globinopathies.Semantic proximity Read ↗

A natural regulatory mutation in the proximal promoter elevates fetal globin expression by creating a de novo GATA1 siteSemantic proximity Read ↗

Differential Binding of the NFE3 and CP1/NFY Transcription Factors to the Human γ- and ∊-Globin CCAAT BoxesSemantic proximity Read ↗

BAP1 deubiquitinase is a potent repressor of fetal hemoglobin biosynthesisSemantic proximity Read ↗

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