Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Temporally distinct myeloid cell responses mediate damage and repair after cerebrovascular injury

18 Jan 2021

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Acute Ischemic Stroke Management
Share to Neuroinflammation and Neurodegeneration Mechanisms
Share to Neurological Disease Mechanisms and Treatments

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 33462481
DOI: 10.1038/s41593-020-00773-6

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Temporally distinct myeloid cell responses mediate damage and repair after cerebrovascular injuryPMID 33462481Read ↗

Phenotypic and spatial heterogeneity of brain myeloid cells after stroke is associated with cell ontogeny, tissue damage, and brain connectivity.Semantic proximity Read ↗

Innate immune memory after brain injury drives inflammatory cardiac dysfunctionSemantic proximity Read ↗

Temporal and Age-Dependent Regulation of Phagocytosis-Related Signatures After Ischemic Stroke: Cross-Species Transcriptomic EvidenceSemantic proximity Read ↗

Novel Therapeutic Strategy Targeting Hspa8 for Ischemic Brain InjurySemantic proximity Read ↗

Update on the role of endothelial cells in trauma.Semantic proximity Read ↗

Microglial immunosurveillance after ischemic stroke: dynamic phenotypes, neurovascular interactions, and therapeutic opportunities.Semantic proximity Read ↗

The Role of Immune Infiltration and Oxidative Stress in the Progression of Cerebral Cavernous Malformation.Semantic proximity Read ↗

Shared inflammatory glial cell signature after brain injury, revealed by spatial, temporal and cell-type-specific profiling of the murine cerebral cortexSemantic proximity Read ↗

Monocyte, Neutrophil and Whole Blood Transcriptome Dynamics Following Ischemic StrokeSemantic proximity Read ↗

Monocyte, neutrophil, and whole blood transcriptome dynamics following ischemic strokeSemantic proximity Read ↗

Cerebrovascular Injuries Induce Lymphatic Invasion into Brain Parenchyma to Guide Vascular Regeneration in ZebrafishSemantic proximity Read ↗

The immunologic phenotype of thrombi is associated with future vascular events after cerebral infarction.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.