Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The New Model of Snail Expression Regulation: The Role of MRTFs in Fast and Slow Endothelial–Mesenchymal Transition

16 Aug 2020

Abstract excerpt

Endothelial–mesenchymal transition (EndMT) is a crucial phenomenon in regulating the development of diseases, including cancer metastasis and fibrotic disorders. The primary regulators of disease development are zinc-finger transcription factors belonging to the Snail family. In this study, we characterized the myocardin-related transcription factor (MRTF)-dependent mechanisms of a human snail promoter regulation...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer Cells and Metastasis
Share to Congenital heart defects research
Share to TGF-β signaling in diseases

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 32824297
DOI: 10.3390/ijms21165875

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The New Model of Snail Expression Regulation: The Role of MRTFs in Fast and Slow Endothelial–Mesenchymal TransitionPMID 32824297Read ↗

Emerging roles of inflammation-mediated endothelial–mesenchymal transition in health and diseaseSemantic proximity Read ↗

TGF-β-Induced Endothelial to Mesenchymal Transition in Disease and Tissue EngineeringSemantic proximity Read ↗

Inhibitory Effect of Essential Oil from Fructus Alpiniae zerumbet on Endothelial-to-Mesenchymal Transformation Induced by TGF-β1 and down-regulating KLF4Semantic proximity Read ↗

Snail Is a Direct Target of Hypoxia-inducible Factor 1α (HIF1α) in Hypoxia-induced Endothelial to Mesenchymal Transition of Human Coronary Endothelial CellsSemantic proximity Read ↗

Fine‐tuning vascular fate during endothelial–mesenchymal transitionSemantic proximity Read ↗

The Role of Snail in EMT and TumorigenesisSemantic proximity Read ↗

The role of endothelial–mesenchymal transition in development and pathological processSemantic proximity Read ↗

Endothelial to Mesenchymal Transition in the Cardiogenesis and Cardiovascular DiseasesSemantic proximity Read ↗

Myocardin-related transcription factor A (MRTF-A) plays an essential role in hepatic stellate cell activation by epigenetically modulating TGF-β signalingSemantic proximity Read ↗

A gene regulatory network to control EMT programs in development and disease.Semantic proximity Read ↗

MicroRNA-31 is a positive modulator of endothelial-mesenchymal transition and associated secretory phenotype induced by TGF-β.Semantic proximity Read ↗

Transcriptional regulation of endothelial-to-mesenchymal transition in cardiac fibrosis: role of myocardin-related transcription factor A and activating transcription factor 3.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.