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Non‐genomic mechanisms in the estrogen regulation of glycolytic protein levels in endothelial cells

5 Aug 2020

Abstract excerpt

Few studies have explored the mechanisms coupling estrogen signals to metabolic demand in endothelial cells. We recently showed that 17β-estradiol (E2) triggers angiogenesis via the membrane G-protein coupled estrogen receptor (GPER) and the key glycolytic protein PFKFB3 as a downstream effector. We herein investigated whether estrogenic agents regulate the stability and/or degradation of glycolytic proteins in...

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PMID: 32757462
DOI: 10.1096/fj.202001130r

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