Article
Smoking-by-genotype interaction in type 2 diabetes risk and fasting glucose.
PloS one - 1 Jan 2020
Wu Peitao, Rybin Denis, Bielak Lawrence F, Feitosa Mary F, Franceschini Nora, Li Yize, Lu Yingchang, Marten Jonathan, Musani Solomon K, Noordam Raymond, Raghavan Sridharan, Rose Lynda M, Schwander Karen, Smith Albert V, Tajuddin Salman M, Vojinovic Dina, Amin Najaf, Arnett Donna K, Bottinger Erwin P, Demirkan Ayse, Florez Jose C, Ghanbari Mohsen, Harris Tamara B, Launer Lenore J, Liu Jingmin, Liu Jun, Mook-Kanamori Dennis O, Murray Alison D, Nalls Mike A, Peyser Patricia A, Uitterlinden André G, Voortman Trudy, Bouchard Claude, Chasman Daniel, Correa Adolfo, de Mutsert Renée, Evans Michele K, Gudnason Vilmundur, Hayward Caroline, Kao Linda, Kardia Sharon L R, Kooperberg Charles, Loos Ruth J F, Province Michael M, Rankinen Tuomo, Redline Susan, Ridker Paul M, Rotter Jerome I, Siscovick David, Smith Blair H, van Duijn Cornelia, Zonderman Alan B, Rao D C, Wilson James G, Dupuis Josée, Meigs James B, Liu Ching-Ti, Vassy Jason L
Abstract excerpt
Smoking is a potentially causal behavioral risk factor for type 2 diabetes (T2D), but not all smokers develop T2D. It is unknown whether genetic factors partially explain this variation. We performed genome-environment-wide interaction studies to identify loci exhibiting potential interaction with baseline smoking status (ever vs. never) on incident T2D and fasting glucose (FG). Analyses were performed in...
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