Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Endothelial NOD1 directs myeloid cell recruitment in atherosclerosis through VCAM‐1

29 Nov 2018

Abstract excerpt

ABSTRACT Atherosclerosis is a chronic disease characterized by vascular lipid retention and inflammation, and pattern recognition receptors (PRRs) are important contributors in early stages of the disease. Given the implication of the intracellular PRR nucleotide‐binding oligomerization domain 1 (NOD1) in cardiovascular diseases, we investigated its contribution to early atherosclerosis. We evidenced NOD1...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Atherosclerosis and Cardiovascular Diseases
Share to Cell Adhesion Molecules Research
Share to Immune Response and Inflammation

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 30496704
DOI: 10.1096/fj.201801231rr

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Endothelial NOD1 directs myeloid cell recruitment in atherosclerosis through VCAM‐1PMID 30496704Read ↗

A major role for RCAN1 in atherosclerosis progression.Semantic proximity Read ↗

Endothelial cell-intrinsic NOD2 signaling regulates the intestinal immune response through the generation of effector and memory T cellsSemantic proximity Read ↗

Synergistic Role of NK Cells and Monocytes in Promoting Atherogenesis in Severe COVID-19 PatientsSemantic proximity Read ↗

Impact of Notch Signaling on Inflammatory Responses in Cardiovascular DisordersSemantic proximity Read ↗

LncRNA VINAS regulates atherosclerosis by modulating NF-κB and MAPK signalingSemantic proximity Read ↗

Thematic Review Series: The Immune System and Atherogenesis. Molecular mechanisms regulating monocyte recruitment in atherosclerosisSemantic proximity Read ↗

ENDOTHELIAL-DEPENDENT MECHANISMS IN CHRONIC INFLAMMATORY LEUKOCYTE RECRUITMENTSemantic proximity Read ↗

Myeloid Type I Interferon Signaling Promotes Atherosclerosis by Stimulating Macrophage Recruitment to LesionsSemantic proximity Read ↗

Regulation of NO Synthesis by Caveolin-1: A Review of Its Importance in Blood Vessels, Perivascular Adipose Tissue and in AtherosclerosisSemantic proximity Read ↗

Leukocyte recruitment in atherosclerosis: Potential targets for therapeutic approaches?Semantic proximity Read ↗

Neutrophil microvesicles drive atherosclerosis by delivering <i>miR-155</i> to atheroprone endotheliumSemantic proximity Read ↗

NR4A1 (Nur77) deletion polarizes macrophages toward an inflammatory phenotype and increases atherosclerosis.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.