Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Congenital Defects in the Expression of the Glycosylphosphatidylinositol-Anchored Complement Regulatory Proteins CD59 and Decay-Accelerating Factor

16 Apr 2018

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Complement system in diseases
Share to Renal Diseases and Glomerulopathies
Share to Trypanosoma species research and implications

Source

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 30032750
DOI: 10.1053/j.seminhematol.2018.04.004

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 10 closest

Congenital Defects in the Expression of the Glycosylphosphatidylinositol-Anchored Complement Regulatory Proteins CD59 and Decay-Accelerating FactorPMID 30032750Read ↗

Congenital CD59 Deficiency.Semantic proximity Read ↗

Association of Blood Group Antigen CD59 with DiseaseSemantic proximity Read ↗

Membrane-bound complement regulator expression in peripheral nervesSemantic proximity Read ↗

CD59a Is the Primary Regulator of Membrane Attack Complex Assembly in the MouseSemantic proximity Read ↗

Crry, but not CD59 and DAF, is indispensable for murine erythrocyte protection in vivo from spontaneous complement attackSemantic proximity Read ↗

Glycosyl phosphatidylinositol-linked blood group antigens and paroxysmal nocturnal hemoglobinuria.Semantic proximity Read ↗

Molecular basis of reduced or absent expression of decay-accelerating factor in Cromer blood group phenotypes.Semantic proximity Read ↗

Expression of complement regulatory proteins CR1, DAF, MCP and CD59in haematological malignanciesSemantic proximity Read ↗

Identification and Functional Characterization of a New Gene Encoding the Mouse Terminal Complement Inhibitor CD59Semantic proximity Read ↗

Mapping the intermedilysin-human CD59 receptor interface reveals a deep correspondence with the binding site on CD59 for complement binding proteins C8alpha and C9.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.