Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Does thinner right entorhinal cortex underlie genetic liability to cannabis use?

Psychological medicine - 1 Dec 2018

Paul Subhadip, Bhattacharyya Sagnik

Abstract excerpt

BACKGROUND: Although alterations in medial temporal lobe structures have been previously associated with use of cannabis, one of the most widely used illicit drugs, whether such alterations are a cause or effect of cannabis use has been unclear. METHODS: In this cross-sectional observational study involving 404 twins/siblings, we have compared cortical thickness and surface area between groups of gender-matched...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cross-Sectional Studies
Share to Entorhinal Cortex
Share to Environment

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genetic Predisposition to Disease

Share to Magnetic Resonance Imaging

Share to Young Adult

Source

Identifiers

PMID: 29482671
DOI: 10.1017/S0033291718000417

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Does thinner right entorhinal cortex underlie genetic liability to cannabis use?PMID 29482671Read ↗

Shared Predisposition in the Association Between Cannabis Use and Subcortical Brain Structure.Semantic proximity Read ↗

Brain Structure and Substance Use: Disentangling Risk, Exposure, and Drug-Specific EffectsSemantic proximity Read ↗

The genetic aetiology of cannabis use: from twin models to genome-wide association studies and beyond.Semantic proximity Read ↗

Early Cannabis Use, Polygenic Risk Score for Schizophrenia and Brain Maturation in Adolescence.Semantic proximity Read ↗

Association of polygenic risk for cannabis use disorder with brain structure among youth with and without bipolar disorder.Semantic proximity Read ↗

Measuring the Associations Between Brain Morphometry and Polygenic Risk Scores for Substance use Disorders in Drug-Naive Adolescents.Semantic proximity Read ↗

Heritability of cortical thickness changes over time in twin pairs discordant for schizophrenia.Semantic proximity Read ↗

Shared genetic effects among schizophrenia, substance use disorder, and hippocampal volume in a multiplex extended pedigree sample.Semantic proximity Read ↗

Effects of environmental risks and polygenic loading for schizophrenia on cortical thicknessSemantic proximity Read ↗

Familial and environmental influences on brain volumes in twins with schizophrenia.Semantic proximity Read ↗

Genetics of cannabis ever-use and frequency across ancestries implicate novel loci and brain-specific biologySemantic proximity Read ↗

Associations between cannabinoid receptor-1 (CNR1) variation and hippocampus and amygdala volumes in heavy cannabis users.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.