Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

[Detection and analysis of phenotypes of tumor-associated macrophages in mouse model of spontaneous breast cancer].

Xi bao yu fen zi mian yi xue za zhi = Chinese journal of cellular and molecular immunology - 1 Jun 2017

Ren Xiaojie, Ren Peng, Luo Song, Ji Quanbo, Xu Meng, Lu Ning, Wang Yan

Abstract excerpt

Objective To detect the phenotypic conversion of tumor-associated macrophages (TAMs) through analyzing the expression levels of the polarization-related genes. Methods We identified the spontaneous breast cancer mice by genotyping and characterized them into the early stage and the advanced stage groups according to their tumor size. Single cell suspension of the tumor tissues were obtained by mechanical methods...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Disease Models, Animal
Share to Interleukin-10

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Interleukin-1beta

Share to Macrophages

Share to Mammary Neoplasms, Experimental

Share to Neoplasm Staging

Share to RNA, Messenger

Source

Identifiers

PMID: 28615091

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

[Detection and analysis of phenotypes of tumor-associated macrophages in mouse model of spontaneous breast cancer].PMID 28615091Read ↗

Class A scavenger receptors promote tumor progression and induce a unique macrophage phenotype in a mouse model of spontaneous breast cancer.Semantic proximity Read ↗

Characterization of Breast Cancer Preclinical Models Reveals a Specific Pattern of Macrophage Polarization.Semantic proximity Read ↗

SAA1 released by tumor cells facilitates the aggressiveness of breast cancer by inducing reprogramming of molecular phenotypes in immune cellsSemantic proximity Read ↗

The cellular and molecular origin of tumor-associated macrophagesSemantic proximity Read ↗

Inhibition of RANK signaling in breast cancer induces an anti-tumor immune response orchestrated by CD8+ T cellsSemantic proximity Read ↗

Comparison of the characteristics of macrophages derived from murine spleen, peritoneal cavity, and bone marrow.Semantic proximity Read ↗

Lactoferrin-containing immunocomplex mediates antitumor effects by resetting tumor-associated macrophages to M1 phenotype.Semantic proximity Read ↗

In situ proliferation contributes to accumulation of tumor‐associated macrophages in spontaneous mammary tumorsSemantic proximity Read ↗

M3 Macrophages Stop Division of Tumor Cells In Vitro and Extend Survival of Mice with Ehrlich Ascites Carcinoma.Semantic proximity Read ↗

Low-dose salinomycin inhibits breast cancer metastasis by repolarizing tumor hijacked macrophages toward the M1 phenotype.Semantic proximity Read ↗

N-glycan-defective breast cancer cells induce a phenotypic switch in polarization of bone marrow-derived macrophages.Semantic proximity Read ↗

Macrophages regulate the angiogenic switch in a mouse model of breast cancer.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.