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Article

A genome-wide analysis of gene-caffeine consumption interaction on basal cell carcinoma.

Carcinogenesis - 1 Dec 2016

Li Xin, Cornelis Marilyn C, Liang Liming, Song Fengju, De Vivo Immaculata, Giovannucci Edward, Tang Jean Y, Han Jiali

Abstract excerpt

Animal models have suggested that oral or topical administration of caffeine could inhibit ultraviolet-induced carcinogenesis via the ataxia telangiectasia and rad3 (ATR)-related apoptosis. Previous epidemiological studies have demonstrated that increased caffeine consumption is associated with reduced risk of basal cell carcinoma (BCC). To identify common genetic markers that may modify this association, we...

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

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Source

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Identifiers

PMID: 27797824
DOI: 10.1093/carcin/bgw107
PMCID: PMC5137266

Genes

NEIL3
ATF3
ATR

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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