Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Genome-Wide Gene Expression Profiling of Randall’s Plaques in Calcium Oxalate Stone Formers

13 Jun 2016

Abstract excerpt

Randall plaques (RPs) can contribute to the formation of idiopathic calcium oxalate (CaOx) kidney stones; however, genes related to RP formation have not been identified. We previously reported the potential therapeutic role of osteopontin (OPN) and macrophages in CaOx kidney stone formation, discovered using genome-recombined mice and genome-wide analyses. Here, to characterize the genetic pathogenesis of RPs,...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Dermatological and Skeletal Disorders
Share to Kidney Stones and Urolithiasis Treatments
Share to Porphyrin Metabolism and Disorders

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 27297950
DOI: 10.1681/asn.2015111271

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Genome-Wide Gene Expression Profiling of Randall’s Plaques in Calcium Oxalate Stone FormersPMID 27297950Read ↗

Construction and Analysis of Immune Infiltration-Related ceRNA Network for Kidney StonesSemantic proximity Read ↗

Is there a pathological switch that triggers the onset of renal calcification?Semantic proximity Read ↗

A spatially anchored transcriptomic atlas of the human kidney papilla identifies significant immune injury and matrix remodeling in patients with stone diseaseSemantic proximity Read ↗

Two distinct phenotypes of calcium oxalate stone formers could imply different long-term risks for renal function.Semantic proximity Read ↗

Recent developments in the study of cellular inflammation in the papillae of stone formers.Semantic proximity Read ↗

A spatially anchored transcriptomic atlas of the human kidney papilla identifies significant immune injury in patients with stone diseaseSemantic proximity Read ↗

Characterizing Chemokine Signaling Pathways and Hub Genes in Calcium Oxalate-Induced Kidney Stone Formation: Insights from Rodent ModelsSemantic proximity Read ↗

Integrated single-nucleus sequencing and spatial architecture analysis identified distinct injured-proximal tubular types in calculi ratsSemantic proximity Read ↗

Identification and validation of the biomarkers related to ferroptosis in calcium oxalate nephrolithiasisSemantic proximity Read ↗

Bioinformatics Analysis of the Cuprotosis Gene in Immune Infiltration of Chronic Kidney DiseaseSemantic proximity Read ↗

Identification of the pivotal role of SPP1 in kidney stone disease based on multiple bioinformatics analysisSemantic proximity Read ↗

Idiopathic calcium nephrolithiasis: a review of pathogenic mechanisms in the light of genetic studies.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.