Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

The epidermal growth factor receptor decreases Stathmin 1 and triggers catagen entry in the mouse.

Experimental dermatology - 1 Apr 2016

Bichsel Kyle J, Hammiller Brianna, Trempus Carol S, Li Yanhua, Hansen Laura A

Abstract excerpt

The epidermal growth factor receptor (EGFR) is necessary for normal involution of hair follicles after the growth phase of anagen, although the mechanisms through which it acts are not well understood. In this report, we used transcriptional profiling of microdissected hair follicles from mice with skin-targeted deletion of Egfr to investigate how EGFR activation triggers catagen. Immunofluorescence for...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Gene Deletion
Share to Chromosomal Proteins, Non-Histone

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to ErbB Receptors

Share to Gene Expression Regulation, Developmental

Share to Hair Follicle

Share to Keratinocytes

Share to Stathmin

Source

Identifiers

PMID: 26661905
DOI: 10.1111/exd.12921

Genes

EGFR

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

The epidermal growth factor receptor decreases Stathmin 1 and triggers catagen entry in the mouse.PMID 26661905Read ↗

Suppression of Wnt/β-catenin signaling by EGF receptor is required for hair follicle development.Semantic proximity Read ↗

Epidermal growth factor as a biologic switch in hair growth cycle.Semantic proximity Read ↗

Expression of a dominant negative mutant of epidermal growth factor receptor in the epidermis of transgenic mice elicits striking alterations in hair follicle development and skin structure.Semantic proximity Read ↗

Runx1 directly promotes proliferation of hair follicle stem cells and epithelial tumor formation in mouse skin.Semantic proximity Read ↗

Beyond wavy hairs: the epidermal growth factor receptor and its ligands in skin biology and pathology.Semantic proximity Read ↗

Development and progression of alopecia in the vitamin D receptor null mouseSemantic proximity Read ↗

Role for the epidermal growth factor receptor in chemotherapy-induced alopecia.Semantic proximity Read ↗

Cutaneous consequences of inhibiting EGF receptor signaling in vivo: normal hair follicle development, but retarded hair cycle induction and inhibition of adipocyte growth in Egfr(Wa5) mice.Semantic proximity Read ↗

Control of murine hair follicle regression (catagen) by TGF‐β1 <i>in vivo</i>Semantic proximity Read ↗

Differential expression of keratin and keratin associated proteins are linked with hair loss condition in spontaneously mutated inbred mice.Semantic proximity Read ↗

Targeted Disruption of Smad4 in Mouse Epidermis Results in Failure of Hair Follicle Cycling and Formation of Skin TumorsSemantic proximity Read ↗

MicroRNA-148a Controls Epidermal and Hair Follicle Stem/Progenitor Cells by Modulating the Activities of ROCK1 and ELF5Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.