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Article

Genotyping of immune-related genetic variants identifies TYK2 as a novel associated locus for idiopathic inflammatory myopathies

9 May 2014

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 24812289
DOI: 10.1136/annrheumdis-2014-205440

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Related research

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Genotyping of immune-related genetic variants identifies TYK2 as a novel associated locus for idiopathic inflammatory myopathiesPMID 24812289Read ↗

Dense genotyping of immune-related loci in idiopathic inflammatory myopathies confirms HLA alleles as the strongest genetic risk factor and suggests different genetic background for major clinical subgroups.Semantic proximity Read ↗

The Genetics of Autoimmune MyositisSemantic proximity Read ↗

Identification of Novel Associations and Localization of Signals in Idiopathic Inflammatory Myopathies Using Genome-Wide Imputation.Semantic proximity Read ↗

Recent advances in the immunogenetics of idiopathic inflammatory myopathy.Semantic proximity Read ↗

Immunogenetics of Idiopathic Inflammatory Myopathies: The Role of HLA Genes Within and Beyond the Ancestral Haplotype.Semantic proximity Read ↗

Strategies for evaluating idiopathic inflammatory myopathy disease susceptibility genes.Semantic proximity Read ↗

Resolving TYK2 locus genotype-to-phenotype differences in autoimmunity.Semantic proximity Read ↗

Discovery of new myositis genetic associations through leveraging other immune-mediated diseases.Semantic proximity Read ↗

Genetic risk and protective factors for the idiopathic inflammatory myopathies.Semantic proximity Read ↗

Risk factors and disease mechanisms in myositis.Semantic proximity Read ↗

Mendelian randomization and clinical trial evidence supports TYK2 inhibition as a therapeutic target for autoimmune diseases.Semantic proximity Read ↗

TYK2 protein-coding variants protect against rheumatoid arthritis and autoimmunity, with no evidence of major pleiotropic effects on non-autoimmune complex traits.Semantic proximity Read ↗

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