Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Adenovirus E1B 19-kilodalton protein modulates innate immunity through apoptotic mimicry.

Journal of virology - 1 Mar 2014

Radke Jay R, Grigera Fernando, Ucker David S, Cook James L

Abstract excerpt

UNLABELLED: Cells that undergo apoptosis in response to chemical or physical stimuli repress inflammatory reactions, but cells that undergo nonapoptotic death in response to such stimuli lack this activity. Whether cells dying from viral infection exhibit a cell death-type modulatory effect on inflammatory reactions is unknown. We compared the effects on macrophage inflammatory responses of cells dying an...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenoviridae
Share to Adenoviridae Infections
Share to Cell Communication

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Cell Death

Share to Cytopathogenic Effect, Viral

Share to Defective Viruses

Share to Inflammation Mediators

Share to Proto-Oncogene Proteins c-bcl-2

Source

Open in PubMed Open full text

Identifiers

PMID: 24352454
DOI: 10.1128/JVI.02372-13
PMCID: PMC3958100

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Adenovirus E1B 19-kilodalton protein modulates innate immunity through apoptotic mimicry.PMID 24352454Read ↗

Identification of FasL as a crucial host factor driving COVID-19 pathology and lethalitySemantic proximity Read ↗

The adenovirus E3/10.4K-14.5K proteins down-modulate the apoptosis receptor Fas/Apo-1 by inducing its internalization.Semantic proximity Read ↗

MAVS-mediated host cell defense is inhibited by Borna disease virus.Semantic proximity Read ↗

Type I interferons are essential mediators of apoptotic death in virally infected cellsSemantic proximity Read ↗

Functional complementation of the adenovirus E1B 19-kilodalton protein with Bcl-2 in the inhibition of apoptosis in infected cellsSemantic proximity Read ↗

The Transcription Factor IRF3 Triggers “Defensive Suicide” Necrosis in Response to Viral and Bacterial PathogensSemantic proximity Read ↗

E1A and E1B proteins inhibit inflammation induced by adenovirus.Semantic proximity Read ↗

Mechanisms of Reovirus-Induced Cell Death and Tissue Injury: Role of Apoptosis and Virus-Induced Perturbation of Host-Cell Signaling and Transcription Factor ActivationSemantic proximity Read ↗

RIPK3 promotes adenovirus type 5 activitySemantic proximity Read ↗

Bid Regulates the Pathogenesis of Neurotropic ReovirusSemantic proximity Read ↗

MAVS-Mediated Apoptosis and Its Inhibition by Viral ProteinsSemantic proximity Read ↗

REGULATORS OF APOPTOSIS ON THE ROAD TO PERSISTENT ALPHAVIRUS INFECTIONSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.