Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

A lovastatin-elicited genetic program inhibits M2 macrophage polarization and enhances T cell infiltration into spontaneous mouse mammary tumors

26 Oct 2013

Abstract excerpt

// Emilia Mira 1 , Lorena Carmona-Rodríguez 1 , Manuel Tardáguila 1 , Iñigo Azcoitia 2 , Alicia González-Martín 1,4 , Luis Almonacid 1 , Josefina Casas 3 , Gemma Fabriás 3 , Santos Mañes 1 1 Department of Immunology and Oncology, Centro Nacional de Biotecnología/CSIC, Madrid, Spain, 2 Department of Cell Biology, School of Biology, Universidad Complutense de Madrid, Madrid, Spain, 3 Department of Biomedicinal...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cancer, Lipids, and Metabolism
Share to Ferroptosis and cancer prognosis
Share to RNA modifications and cancer

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Source

Identifiers

PMID: 24317954
DOI: 10.18632/oncotarget.1376

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

A lovastatin-elicited genetic program inhibits M2 macrophage polarization and enhances T cell infiltration into spontaneous mouse mammary tumorsPMID 24317954Read ↗

Functional Genetic Screens Reveal Key Pathways Instructing the Molecular Phenotypes of Tumor-Associated MacrophagesSemantic proximity Read ↗

In situ proliferation contributes to accumulation of tumor‐associated macrophages in spontaneous mammary tumorsSemantic proximity Read ↗

Characterization of Breast Cancer Preclinical Models Reveals a Specific Pattern of Macrophage Polarization.Semantic proximity Read ↗

Inflammation and stem markers association to PIM1/PIM2 kinase-induced tumors in breast and uterusSemantic proximity Read ↗

Macrophages regulate the angiogenic switch in a mouse model of breast cancer.Semantic proximity Read ↗

Mammary carcinoma cell derived cyclooxygenase 2 suppresses tumor immune surveillance by enhancing intratumoral immune checkpoint activity.Semantic proximity Read ↗

MicroRNA-200c restoration reveals a cytokine profile to enhance M1 macrophage polarization in breast cancerSemantic proximity Read ↗

Wnt Signaling in the Phenotype and Function of Tumor-Associated Macrophages.Semantic proximity Read ↗

Sphingosine kinase 2 deficient tumor xenografts show impaired growth and fail to polarize macrophages towards an anti-inflammatory phenotype.Semantic proximity Read ↗

Endostatin inhibits the growth and migration of 4T1 mouse breast cancer cells by skewing macrophage polarity toward the M1 phenotype.Semantic proximity Read ↗

ErbB2 Transgenic Mice: A Tool for Investigation of the Immune Prevention and Treatment of Mammary CarcinomasSemantic proximity Read ↗

CD4(+) T cells regulate pulmonary metastasis of mammary carcinomas by enhancing protumor properties of macrophages.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.