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Article

Transcriptional activation of transposable elements in mouse zygotes is independent of Tet3-mediated 5-methylcytosine oxidation

27 Nov 2012

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Share to Epigenetics and DNA Methylation
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 23184059
DOI: 10.1038/cr.2012.160

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Related research

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Research trail→

Click a neighbor to travelStep 1 · 12 closest

Transcriptional activation of transposable elements in mouse zygotes is independent of Tet3-mediated 5-methylcytosine oxidationPMID 23184059Read ↗

Maternal TET3 is dispensable for embryonic development but is required for neonatal growthSemantic proximity Read ↗

Regulatory changes as a consequence of transposon insertion.Semantic proximity Read ↗

Reprogramming of the paternal genome upon fertilization involves genome-wide oxidation of 5-methylcytosineSemantic proximity Read ↗

Epigenetic reprogramming by TET enzymes impacts co-transcriptional R-loopsSemantic proximity Read ↗

Transposable elements in cancer and other human diseases.Semantic proximity Read ↗

Epigenetics. Satellite tools.Semantic proximity Read ↗

Epigenetic reprogramming in early mammalian development and following somatic nuclear transferSemantic proximity Read ↗

Transposable elements and the epigenetic regulation of the genome.Semantic proximity Read ↗

Functionally distinct roles for TET-oxidized 5-methylcytosine bases in somatic reprogramming to pluripotency.Semantic proximity Read ↗

The role of Tet3 DNA dioxygenase in epigenetic reprogramming by oocytes.Semantic proximity Read ↗

OGT prevents DNA demethylation and suppresses the expression of transposable elements in heterochromatin by restraining TET activity genome-wideSemantic proximity Read ↗

Historical overview of transposable element research.Semantic proximity Read ↗

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