Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Development of an autism severity score for mice using Nlgn4 null mutants as a construct-valid model of heritable monogenic autism.

Behavioural brain research - 15 Aug 2013

El-Kordi Ahmed, Winkler Daniela, Hammerschmidt Kurt, Kästner Anne, Krueger Dilja, Ronnenberg Anja, Ritter Caroline, Jatho Jasmin, Radyushkin Konstantin, Bourgeron Thomas, Fischer Julia, Brose Nils, Ehrenreich Hannelore

Abstract excerpt

Autism is the short name of a complex and heterogeneous group of disorders (autism spectrum disorders, ASD) with several lead symptoms required for classification, including compromised social interaction, reduced verbal communication and stereotyped repetitive behaviors/restricted interests. The etiology of ASD is still unknown in most cases but monogenic heritable forms exist that have provided insights into...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Autistic Disorder
Share to Behavior, Animal

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Cell Adhesion Molecules, Neuronal

Share to Grooming

Share to Sex Characteristics

Share to Social Behavior

Share to Stereotyped Behavior

Share to Vocalization, Animal

Source

Identifiers

PMID: 23183221
DOI: 10.1016/j.bbr.2012.11.016

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 3 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Development of an autism severity score for mice using Nlgn4 null mutants as a construct-valid model of heritable monogenic autism.PMID 23183221Read ↗

Juvenile manifestation of ultrasound communication deficits in the neuroligin-4 null mutant mouse model of autism.Semantic proximity Read ↗

Absence of deficits in social behaviors and ultrasonic vocalizations in later generations of mice lacking neuroligin4.Semantic proximity Read ↗

Developmental delays and reduced pup ultrasonic vocalizations but normal sociability in mice lacking the postsynaptic cell adhesion protein neuroligin2.Semantic proximity Read ↗

Comprehensive Analysis of the 16p11.2 Deletion and Null Cntnap2 Mouse Models of Autism Spectrum Disorder.Semantic proximity Read ↗

Neuroligin‐3‐deficient mice: model of a monogenic heritable form of autism with an olfactory deficitSemantic proximity Read ↗

Juvenile Shank3b deficient mice present with behavioral phenotype relevant to autism spectrum disorder.Semantic proximity Read ↗

Behavioral profiles of mouse models for autism spectrum disorders.Semantic proximity Read ↗

Advances in behavioral genetics: mouse models of autism.Semantic proximity Read ↗

Behavioural Phenotypes and Neural Circuit Dysfunctions in Mouse Models of Autism Spectrum Disorder.Semantic proximity Read ↗

Twenty years of discoveries emerging from mouse models of autism.Semantic proximity Read ↗

Behavioral phenotypes of genetic mouse models of autism.Semantic proximity Read ↗

Abnormal social behaviors and dysfunction of autism-related genes associated with daily agonistic interactions in miceSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.