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Article

Contribution of SAM and HD domains to retroviral restriction mediated by human SAMHD1

13 Nov 2012

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 23158101
DOI: 10.1016/j.virol.2012.10.029

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Related research

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Contribution of SAM and HD domains to retroviral restriction mediated by human SAMHD1PMID 23158101Read ↗

How Samhd1 changes our view of viral restrictionSemantic proximity Read ↗

Evolutionary and Functional Analyses of the Interaction between the Myeloid Restriction Factor SAMHD1 and the Lentiviral Vpx ProteinSemantic proximity Read ↗

SAMHD1 suppresses innate immune responses to viral infections and inflammatory stimuli by inhibiting the NF-κB and interferon pathwaysSemantic proximity Read ↗

Low dNTP levels are necessary but may not be sufficient for lentiviral restriction by SAMHD1Semantic proximity Read ↗

Phosphorylation of murine SAMHD1 regulates its antiretroviral activitySemantic proximity Read ↗

Restriction of diverse retroviruses by SAMHD1Semantic proximity Read ↗

Effects of human SAMHD1 polymorphisms on HIV-1 susceptibility.Semantic proximity Read ↗

CD81 association with SAMHD1 enhances HIV-1 reverse transcription by increasing dNTP levelsSemantic proximity Read ↗

SAMHD1 restricts HIV-1 infection in resting CD4+ T cellsSemantic proximity Read ↗

Human cytomegalovirus overcomes SAMHD1 restriction in macrophages via pUL97Semantic proximity Read ↗

SAMHD1-mediated HIV-1 restriction in cells does not involve ribonuclease activitySemantic proximity Read ↗

AIDS/HIV. HIV interplay with SAMHD1.Semantic proximity Read ↗

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