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Article

Mechanisms associated to impaired activity of cardiac P-type ATPases in endothelial nitric oxide synthase knockout mice.

Journal of physiology and biochemistry - 1 Jun 2013

Rezende Daniele C, Pôças Elisa S C, Muzi-Filho Humberto, Cunha Valéria M N, Caricati-Neto Afonso, Jurkiewicz Aron, Noël François, Quintas Luis E M

Abstract excerpt

The effect of long-lasting in vivo restriction of nitric oxide (NO) bioavailability on cardiac and renal P-type ATPases critical for intracellular ion homeostasis is controversial. Previous work has shown in eNOS knockout (eNOS(-/-)) mice hearts that Na(+)/K(+)- and Ca(2+)-ATPase activities were depressed but the underlying mechanisms are still unclear. The goal of this study was to characterize potential...

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PMID: 23011780
DOI: 10.1007/s13105-012-0203-x

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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