Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Identification of a novel partner gene, <i>TPR</i>, fused to <i>FGFR1</i> in 8p11 myeloproliferative syndrome

23 May 2012

Abstract excerpt

The 8p11 myeloproliferative syndrome (EMS) is an aggressive neoplasm caused by the fusion of various partner genes to fibroblast growth factor receptor 1 (FGFR1). Various FGFR1 fusions are associated with subtly distinct disease phenotypes. Here, we report a new translocation at the FGFR1 locus in a patient who carried t(1;8)(q25;p11.2) and presented with myeloproliferative neoplasm-like symptoms. The patient was...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Eosinophilic Disorders and Syndromes
Share to Histiocytic Disorders and Treatments
Share to Myeloproliferative Neoplasms: Diagnosis and Treatment

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Source

Identifiers

PMID: 22619110
DOI: 10.1002/gcc.21973

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 24 Oct 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Identification of a novel partner gene, <i>TPR</i>, fused to <i>FGFR1</i> in 8p11 myeloproliferative syndromePMID 22619110Read ↗

Identification of a novel gene, <i>FGFR1OP2</i>, fused to <i>FGFR1</i> in 8p11 myeloproliferative syndromeSemantic proximity Read ↗

8p11 myeloproliferative syndrome with a novel t(7;8) translocation leading to fusion of the FGFR1 and TIF1 genes.Semantic proximity Read ↗

LRRFIP1, a new FGFR1 partner gene associated with 8p11 myeloproliferative syndromeSemantic proximity Read ↗

Fusion gene‐mediated truncation of <i>RUNX1</i> as a potential mechanism underlying disease progression in the 8p11 myeloproliferative syndromeSemantic proximity Read ↗

Fusion of the <i>BCR</i> and the fibroblast growth factor receptor‐1 (<i>FGFR1</i>) genes as a result of t(8;22)(p11;q11) in a myeloproliferative disorder: The first fusion gene involving <i>BCR</i> but not <i>ABL</i>Semantic proximity Read ↗

The 8p11 Myeloproliferative Syndrome: A Distinct Clinical Entity Caused by Constitutive Activation of <i>FGFR1</i>Semantic proximity Read ↗

Myeloid/lymphoid neoplasms with FGFR1 rearrangement.Semantic proximity Read ↗

The 8p11 myeloproliferative syndrome: Genotypic and phenotypic classification and targeted therapySemantic proximity Read ↗

Myeloid and lymphoid neoplasms with FGFR1 abnormalities: diagnostic and therapeutic challenges.Semantic proximity Read ↗

The t(6;8)(q27;p11) Translocation in a Stem Cell Myeloproliferative Disorder Fuses a Novel Gene, FOP, to Fibroblast Growth Factor Receptor 1Semantic proximity Read ↗

FGFR1 is fused to the centrosome-associated proteinCEP110 in the 8p12 stem cell myeloproliferative disorder with t(8;9)(p12;q33)Semantic proximity Read ↗

Myeloid and lymphoid neoplasm with FGFR1 abnormality.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.