CD14 cooperates with complement receptor 3 to mediate MyD88-independent phagocytosis of <i>Borrelia burgdorferi</i>

Abstract excerpt

Phagocytosis of Borrelia burgdorferi, the causative agent of Lyme disease, is a poorly understood process, despite its importance during the host immune response to infection. B. burgdorferi has been shown to bind to different receptors on the surface of phagocytic cells, including the β(2) integrin, complement receptor 3 (CR3). However, whether these receptors mediate the phagocytosis of the spirochete remains...

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