Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Adverse host factors exacerbate occult HIV-associated nephropathy.

The American journal of pathology - 1 Oct 2011

Kumar Dileep, Salhan Divya, Magoon Sandeep, Torri Deepti D, Sayeneni Swapna, Sagar Ankita, Bandhlish Anshu, Malhotra Ashwani, Chander Praveen N, Singhal Pravin C

Abstract excerpt

In the present study, we hypothesized that HIV-1-induced occult HIV-associated nephropathy (HIVAN) would become apparent in the presence of adverse host factors. To test our hypothesis, Vpr mice (which display doxycycline-dependent Vpr expression in podocytes) with two, three, and four copies of the angiotensinogen (Agt) gene (Vpr-Agt-2, Vpr-Agt-3, and Vpr-Agt-4) were administered doxycycline for 3 weeks (to...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Angiotensin II
Share to Angiotensinogen

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Connective Tissue Growth Factor

Share to Doxycycline

Share to Epithelial-Mesenchymal Transition

Share to Proteinuria

Share to Renin-Angiotensin System

Source

Open in PubMed Open full text

Identifiers

PMID: 21871425
DOI: 10.1016/j.ajpath.2011.06.013
PMCID: PMC3181397

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Adverse host factors exacerbate occult HIV-associated nephropathy.PMID 21871425Read ↗

Transgenic and Infectious Animal Models of HIV-Associated NephropathySemantic proximity Read ↗

Genetic susceptibility, HIV infection, and the kidney.Semantic proximity Read ↗

Advances in the pathogenesis of HIV-associated kidney diseases.Semantic proximity Read ↗

The Pathogenesis of HIV-Associated NephropathySemantic proximity Read ↗

HIV-associated nephropathy: pathogenesisSemantic proximity Read ↗

Pathogenesis of HIV-associated nephropathy in children and adolescents: taking a hard look 40 years later in the era of gene-environment interactions.Semantic proximity Read ↗

Modulation of renin angiotensin system predominantly alters sclerotic phenotype of glomeruli in HIVAN.Semantic proximity Read ↗

Kidney disease in the setting of HIV infection: conclusions from a Kidney Disease: Improving Global Outcomes (KDIGO) Controversies Conference.Semantic proximity Read ↗

Angiotensin II provokes podocyte injury in murine model of HIV-associated nephropathy.Semantic proximity Read ↗

HIV-associated nephropathy in African Americans11The content of this publication does not necessarily reflect the views or policies of the Department of Health and Human Services, nor does mention of trade names, commercial products, or organizations imply endorsement by the U.S. Government. The publisher or recipient acknowledges right of the U.S. Government to retain a nonexclusive, royalty-free license in and to any copyright covering the article.Semantic proximity Read ↗

The molecular pathogenesis of HIV-1 associated nephropathy: recent advances.Semantic proximity Read ↗

Expression of HIV-1 genes in podocytes alone can lead to the full spectrum of HIV-1-associated nephropathy.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.