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Article

Lte1 promotes mitotic exit by controlling the localization of the spindle position checkpoint kinase Kin4.

Proceedings of the National Academy of Sciences of the United States of America - 2 Aug 2011

Falk Jill E, Chan Leon Y, Amon Angelika

Abstract excerpt

For a daughter cell to receive a complete genomic complement, it is essential that the mitotic spindle be positioned accurately within the cell. In budding yeast, a signaling system known as the spindle position checkpoint (SPOC) monitors spindle position and regulates the activity of the mitotic exit network (MEN), a GTPase signaling pathway that promotes exit from mitosis. The protein kinase Kin4 is a central...

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Identifiers

PMID: 21709215
DOI: 10.1073/pnas.1107784108
PMCID: PMC3150932

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

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