Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Endothelial nitric oxide synthase T-786C mutation, a reversible etiology of Prinzmetal's angina pectoris.

The American journal of cardiology - 15 Mar 2010

Glueck Charles J, Munjal Jitender, Khan Ayub, Umar Muhammad, Wang Ping

Abstract excerpt

Because the endothelial nitric oxide synthase (eNOS) T-786C polymorphism is associated with reduced nitric oxide production and coronary artery spasm in Japanese patients, we speculated that it might be reversibly associated with Prinzmetal's variant angina in white Americans. Polymerase chain reaction analyses of eNOS T-786C and stromelysin 5A6A polymorphisms were done in 31 women and 12 men (42 white and 1...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Administration, Oral
Share to Arginine
Share to Gene Frequency

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Heterozygote

Share to Homozygote

Share to Matrix Metalloproteinase 3

Share to Nitric Oxide

Share to Nitric Oxide Synthase Type III

Source

Identifiers

PMID: 20211321
DOI: 10.1016/j.amjcard.2009.10.062

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Endothelial nitric oxide synthase T-786C mutation, a reversible etiology of Prinzmetal's angina pectoris.PMID 20211321Read ↗

The endothelial nitric oxide synthase T-786c mutation, a treatable etiology of Prinzmetal's angina.Semantic proximity Read ↗

A T-786-->C mutation in the 5'-flanking region of the endothelial nitric oxide synthase gene and coronary arterial vasomotility.Semantic proximity Read ↗

T786C Mutation in the Endothelial Nitric Oxide Synthase Gene in Patients With Primary Osteonecrosis.Semantic proximity Read ↗

T(-786)--> C mutation in the 5'-flanking region of the endothelial nitric oxide synthase gene is associated with myocardial infarction, especially without coronary organic stenosis.Semantic proximity Read ↗

The T-786C endothelial nitric oxide synthase genotype is a novel risk factor for coronary artery disease in Caucasian patients of the GENICA study.Semantic proximity Read ↗

Association of endothelial nitric oxide synthase promoter region (T-786C) gene polymorphism with acute coronary syndrome and coronary heart disease.Semantic proximity Read ↗

Nitric oxide synthase gene G298 allele. Is it a marker for microvascular angina in hypertensive patients?Semantic proximity Read ↗

The T(-786)C endothelial nitric oxide synthase genotype predicts cardiovascular mortality in high-risk patients.Semantic proximity Read ↗

The T-786-->C mutation in endothelial nitric oxide synthase is associated with hypertension.Semantic proximity Read ↗

The endothelial nitric oxide synthase gene -786T/C polymorphism is a predictive factor for reattacks of coronary spasm.Semantic proximity Read ↗

The relationship between endothelial nitric oxide synthase gene polymorphism (T-786 C) and coronary artery disease in the Turkish population.Semantic proximity Read ↗

Synergistic interaction of T-786-->C polymorphism in the endothelial nitric oxide synthase gene and smoking for an enhanced risk for coronary spasm.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.