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Article

Atp10a, a gene adjacent to the PWS/AS gene cluster, is not imprinted in mouse and is insensitive to the PWS-IC

5 Nov 2009

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Share to Epigenetics and DNA Methylation
Share to Genetic Syndromes and Imprinting
Share to Prenatal Screening and Diagnostics

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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Identifiers

PMID: 19894069
DOI: 10.1007/s10048-009-0226-9

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Related research

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Atp10a, a gene adjacent to the PWS/AS gene cluster, is not imprinted in mouse and is insensitive to the PWS-ICPMID 19894069Read ↗

Atp10a, the mouse ortholog of the human imprinted ATP10A gene, escapes genomic imprinting.Semantic proximity Read ↗

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Control elements within the PWS/AS imprinting box and their function in the imprinting process.Semantic proximity Read ↗

Deficiency of Rbbp1/Arid4a and Rbbp1l1/Arid4b alters epigenetic modifications and suppresses an imprinting defect in the PWS/AS domain.Semantic proximity Read ↗

Regulatory elements associated with paternally-expressed genes in the imprinted murine Angelman/Prader-Willi syndrome domain.Semantic proximity Read ↗

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Genomic organization and characterization of mouse SAP , the gene that is altered in X-linked lymphoproliferative diseaseSemantic proximity Read ↗

Mouse imprinting defect mutations that model Angelman syndrome.Semantic proximity Read ↗

Transcription is required to establish maternal imprinting at the Prader-Willi syndrome and Angelman syndrome locus.Semantic proximity Read ↗

A novel maternally expressed gene, ATP10C, encodes a putative aminophospholipid translocase associated with Angelman syndrome.Semantic proximity Read ↗

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