Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Characterization of Borrelia burgdorferi aggregates.

Vector borne and zoonotic diseases (Larchmont, N.Y.) - 1 Jun 2009

Srivastava Siddharth Y, de Silva Aravinda M

Abstract excerpt

Lyme disease is caused by the tick-borne spirochete, Borrelia burgdorferi. It has been documented that B. burgdorferi form aggregates within ticks and during in vitro culture. However, Borrelia aggregates remain poorly characterized, and their functional significance is unknown. Here we have characterized Borrelia aggregates using microscopy and flow cytometry. Borrelia aggregation was temperature, pH, and growth...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Bacterial Adhesion
Share to Bacterial Proteins
Share to Borrelia burgdorferi

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Flow Cytometry

Share to Gene Expression Regulation, Bacterial

Share to Hydrogen-Ion Concentration

Share to Mutation

Share to Temperature

Source

Open in PubMed Open full text

Identifiers

PMID: 19499997
DOI: 10.1089/vbz.2008.0148
PMCID: PMC2904187

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Characterization of Borrelia burgdorferi aggregates.PMID 19499997Read ↗

Expression of <i>Borrelia burgdorferi</i> OspC and DbpA is controlled by a RpoN–RpoS regulatory pathwaySemantic proximity Read ↗

RpoS is not central to the general stress response in Borrelia burgdorferi but does control expression of one or more essential virulence determinants.Semantic proximity Read ↗

Transcriptomic insights on the virulence-controlling CsrA, BadR, RpoN, and RpoS regulatory networks in the Lyme disease spirochete.Semantic proximity Read ↗

Activation of the RpoN-RpoS regulatory pathway during the enzootic life cycle of Borrelia burgdorferiSemantic proximity Read ↗

Who is the BosR around here anyway?Semantic proximity Read ↗

Proteomic Analyses of Morphological Variants of <i>Borrelia burgdorferi</i> Shed New Light on Persistence Mechanisms: Implications for Pathogenesis, Diagnosis and TreatmentSemantic proximity Read ↗

The RpoS Gatekeeper in Borrelia burgdorferi: An Invariant Regulatory Scheme That Promotes Spirochete Persistence in Reservoir Hosts and Niche DiversitySemantic proximity Read ↗

DksA-dependent regulation of RpoS contributes to <i>Borrelia burgdorferi</i> tick-borne transmission and mammalian infectivitySemantic proximity Read ↗

Abrogation of ospAB constitutively activates the Rrp2-RpoN-RpoS pathway (sigmaN-sigmaS cascade) in Borrelia burgdorferi.Semantic proximity Read ↗

Coupled induction of prophage and virulence factors during tick transmission of the Lyme disease spirocheteSemantic proximity Read ↗

Borrelia burgdorferi bba74Is Expressed Exclusively during Tick Feeding and Is Regulated by Both Arthropod- and Mammalian Host-Specific SignalsSemantic proximity Read ↗

Analysis of the RpoS regulon in <i>Borrelia burgdorferi</i> in response to mammalian host signals provides insight into RpoS function during the enzootic cycleSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.