Redundant and synergistic mechanisms control the sequestration of blood-born adenovirus in the liver.

doi:10.1038/mt.2008.307

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Community

Back to search

Article

Redundant and synergistic mechanisms control the sequestration of blood-born adenovirus in the liver.

Molecular therapy : the journal of the American Society of Gene Therapy - 1 Apr 2009

Di Paolo Nelson C, van Rooijen Nico, Shayakhmetov Dmitry M

Abstract excerpt

Human adenovirus (Ad) is a ubiquitous pathogen causing a wide range of diseases. Although the interactions of human Ad serotype 5 (Ad5) with susceptible cells in vitro are known in great detail, host factors controlling the tissue specificity of Ad5 infection in vivo remain poorly understood. Here, we analyzed the mechanisms of sequestration by the liver for blood-born human Ads and Ad5-based vectors. Our data...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Adenoviridae
Share to Capsid Proteins
Share to Factor X
Share to Kupffer Cells

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Liver

Share to Oligopeptides

Share to Transduction, Genetic

Share to Viral Proteins

Source

Open in PubMed Open full text

Identifiers

PMID: 19223863
DOI: 10.1038/mt.2008.307
PMCID: PMC2835106

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.