Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Reactive oxygen species activate HIV long terminal repeat via post-translational control of NF-kappaB.

Biochemical and biophysical research communications - 7 Nov 2008

Pyo Chul-Woong, Yang Young Lae, Yoo Na-Kyung, Choi Sang-Yun

Abstract excerpt

Reduction/oxidation disorder is one of the most common ailments in HIV-infected patients, and such patients are frequently left exposed to chronic oxidative stress after the generation of reactive oxygen species (ROS). Although a variety of clinical trials to inhibit HIV infection have been conducted by focusing on oxidative stress, their precise targets and reaction mechanism have remained unclear. In this...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Acetylation
Share to Gene Expression Regulation, Viral

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to HIV Long Terminal Repeat

Share to I-kappa B Proteins

Share to Jurkat Cells

Share to Luciferases

Share to p300-CBP Transcription Factors

Share to Transcription Factor RelA

Source

Identifiers

PMID: 18765232
DOI: 10.1016/j.bbrc.2008.08.114

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Reactive oxygen species activate HIV long terminal repeat via post-translational control of NF-kappaB.PMID 18765232Read ↗

The generation of ROS by exposure to trihalomethanes promotes the IκBα/NF-kB/p65 complex dissociation in human lung fibroblastSemantic proximity Read ↗

Nuclear Factor Kb: An Oxidative Stress-Responsive Transcription Factor of Eukaryotic Cells (A Review)Semantic proximity Read ↗

HIV-1 Tat protein induces DNA damage in human peripheral blood B-lymphocytes via mitochondrial ROS productionSemantic proximity Read ↗

Antioxidant nanozyme counteracts HIV-1 by modulating intracellular redox potentialSemantic proximity Read ↗

Nuclear Factor Kappa B: A Potential Target for Anti-HIV ChemotherapySemantic proximity Read ↗

Role of free radicals in viral pathogenesis and mutation.Semantic proximity Read ↗

The endogenous reactive oxygen species promote NF-kappaB activation by targeting on activation of NF-kappaB-inducing kinase in oral squamous carcinoma cells.Semantic proximity Read ↗

ROS upregulation during the early phase of retroviral infection plays an important role in viral establishment in the host cell.Semantic proximity Read ↗

Activator protein 1 (AP-1)- and nuclear factor kappaB (NF-kappaB)-dependent transcriptional events in carcinogenesis.Semantic proximity Read ↗

Hydrogen sulfide blocks HIV rebound by maintaining mitochondrial bioenergetics and redox homeostasisSemantic proximity Read ↗

HIV-1 tat expression and sulphamethoxazole hydroxylamine mediated oxidative stress alter the disulfide proteome in Jurkat T cells.Semantic proximity Read ↗

c-Jun NH2-terminal kinase-mediated redox-dependent degradation of IkappaB: role of thioredoxin in NF-kappaB activation.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.