Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

DA rats from two colonies differ genetically and in their arthritis susceptibility.

Mammalian genome : official journal of the International Mammalian Genome Society - 1 Jun 2008

Rintisch Carola, Holmdahl Rikard

Abstract excerpt

The arthritis-susceptible DA rat is one of the most commonly used rat strains for genetic linkage analysis and is instrumental for the identification of many genetic loci. Even though DA rats were kept as inbred lines at different institutes and suppliers, it became obvious that the various breeding stocks differed genetically. To be able to compare the results from different linkage studies it is very import to...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Arthritis
Share to Arthritis, Experimental
Share to Chromosomes, Mammalian

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Genetic Predisposition to Disease

Share to Genome

Share to Physical Chromosome Mapping

Share to Rats, Inbred Strains

Source

Open in PubMed Open full text

Identifiers

PMID: 18668290
DOI: 10.1007/s00335-008-9125-x
PMCID: PMC7088020

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

DA rats from two colonies differ genetically and in their arthritis susceptibility.PMID 18668290Read ↗

Mapping and functional characterization of rat chromosome 4 regions that regulate arthritis models and phenotypes in congenic strains.Semantic proximity Read ↗

Quest for arthritis-causative genetic factors in the rat.Semantic proximity Read ↗

Identification and isolation of dominant susceptibility loci for pristane-induced arthritis.Semantic proximity Read ↗

Rheumatoid arthritis: identifying and characterising polymorphisms using rat models.Semantic proximity Read ↗

Both common and unique susceptibility genes in different rat strains with pristane-induced arthritis.Semantic proximity Read ↗

An integrated genetic linkage map with 1,137 markers constructed from five F2 crosses of autoimmune disease-prone and -resistant inbred rat strains.Semantic proximity Read ↗

Detection of arthritis-susceptibility loci, including Ncf1, and variable effects of the major histocompatibility complex region depending on genetic background in rats.Semantic proximity Read ↗

Modulation of multiple experimental arthritis models by collagen‐induced arthritis quantitative trait loci isolated in congenic rat lines: Different effects of non–major histocompatibility complex quantitative trait loci in males and femalesSemantic proximity Read ↗

Genetic analysis of collagen-induced arthritis in rats: a polygenic model for rheumatoid arthritis predicts a common framework of cross-species inflammatory/autoimmune disease loci.Semantic proximity Read ↗

Contribution of genetic studies in rodent models of autoimmune arthritis to understanding and treatment of rheumatoid arthritis.Semantic proximity Read ↗

The Non-MHC Quantitative Trait Locus Cia5 Contains Three Major Arthritis Genes That Differentially Regulate Disease Severity, Pannus Formation, and Joint Damage in Collagen- and Pristane-Induced ArthritisSemantic proximity Read ↗

Identification of candidate risk gene variations by whole-genome sequence analysis of four rat strains commonly used in inflammation research.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.