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Article

An equine infectious anemia virus variant superinfects cells through novel receptor interactions.

Journal of virology - 1 Oct 2008

Brindley Melinda A, Zhang Baoshan, Montelaro Ronald C, Maury Wendy

Abstract excerpt

Wild-type strains of equine infectious anemia virus (EIAV) prevent superinfection of previously infected cells. A variant strain of virus that spontaneously arose during passage, EIAV(vMA-1c), can circumvent this mechanism in some cells, such as equine dermis (ED) cells, but not in others, such as equine endothelial cells. EIAV(vMA-1c) superinfection of ED cells results in a buildup of unintegrated viral DNA and...

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Source

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Identifiers

PMID: 18667522
DOI: 10.1128/JVI.01142-08
PMCID: PMC2546952

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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