Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Role of polymorphonuclear neutrophils on infectious complications stemming from Enterococcus faecalis oral infection in thermally injured mice.

Journal of immunology (Baltimore, Md. : 1950) - 15 Mar 2008

Tsuda Yasuhiro, Shigematsu Kenji, Kobayashi Makiko, Herndon David N, Suzuki Fujio

Abstract excerpt

Thermally injured mice are susceptible to Enterococcus faecalis translocation. In this study, the role of polymorphonuclear neutrophils (PMN) on the development of sepsis stemming from E. faecalis translocation was studied in SCID-beige (SCIDbg) mice depleted of PMN (SCIDbgN mice) or macrophages (Mphi) and PMN (SCIDbgMN mice). Sepsis was not developed in SCIDbgN mice orally infected with E. faecalis, while the...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Burns
Share to Enterococcus faecalis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Gram-Positive Bacterial Infections

Share to Macrophage Activation

Share to Mice, SCID

Share to Mouth Diseases

Share to Neutropenia

Share to Neutrophils

Source

Identifiers

PMID: 18322224
DOI: 10.4049/jimmunol.180.6.4133

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Role of polymorphonuclear neutrophils on infectious complications stemming from Enterococcus faecalis oral infection in thermally injured mice.PMID 18322224Read ↗

Aberrant inflammatory response to Streptococcus pyogenes in mice lacking myeloid differentiation factor 88.Semantic proximity Read ↗

Neutrophils in innate immunity and systems biology‐level approachesSemantic proximity Read ↗

The role of neutrophil typing in homeostatic resilience protective model of inflammatory preconditioningSemantic proximity Read ↗

Gamma Interferon Does Not Enhance Clearance ofPseudomonas aeruginosabut Does Amplify a Proinflammatory Response in a Murine Model of Postseptic ImmunosuppressionSemantic proximity Read ↗

α-Toxin Regulates Local Granulocyte Expansion from Hematopoietic Stem and Progenitor Cells in Staphylococcus aureus-Infected Wounds.Semantic proximity Read ↗

M2b Monocytes Provoke Bacterial Pneumonia and Gut Bacteria-Associated Sepsis in Alcoholics.Semantic proximity Read ↗

Defect in neutrophil killing and increased susceptibility to infection with nonpathogenic gram-positive bacteria in peptidoglycan recognition protein-S (PGRP-S)-deficient mice.Semantic proximity Read ↗

Myeloid arginase-1 controls excessive inflammation and modulates T cell responses in Pseudomonas aeruginosa pneumonia.Semantic proximity Read ↗

TLR4 influences the humoral and cellular immune response during polymicrobial sepsis.Semantic proximity Read ↗

Epithelial and neutrophil interactions and coordinated response to <i>Shigella</i> in a human intestinal enteroid-neutrophil co-culture modelSemantic proximity Read ↗

Neutrophil Phenotypes and Functions in Trauma and Trauma-Related Sepsis.Semantic proximity Read ↗

Spontaneous neutrophil migration patterns during sepsis after major burns.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.