Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Mutations in the Drosophila mitochondrial tRNA amidotransferase, bene/gatA, cause growth defects in mitotic and endoreplicating tissues.

Genetics - 1 Feb 2008

Morris Jason Z, Bergman Leah, Kruyer Anna, Gertsberg Mikhail, Guigova Adriana, Arias Ronald, Pogorzelska Monika

Abstract excerpt

Rapid larval growth is essential in the development of most metazoans. In this article, we show that bene, a gene previously identified on the basis of its oogenesis defects, is also required for larval growth and viability. We show that all bene alleles disrupt gatA, which encodes the Drosophila homolog of glutamyl-tRNA(Gln) amidotransferase subunit A (GatA). bene alleles are now referred to as gatA. GatA...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Drosophila
Share to Drosophila Proteins

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Mitochondria

Share to Mitosis

Share to Mutation

Share to Polymerase Chain Reaction

Share to Salivary Glands

Source

Open in PubMed Open full text

Identifiers

PMID: 18245325
DOI: 10.1534/genetics.107.084376
PMCID: PMC2248357

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Mutations in the Drosophila mitochondrial tRNA amidotransferase, bene/gatA, cause growth defects in mitotic and endoreplicating tissues.PMID 18245325Read ↗

An extensive allelic series of Drosophila kae1 mutants reveals diverse and tissue-specific requirements for t6A biogenesis.Semantic proximity Read ↗

Drosophila variable nurse cells encodes arrest defective 1 (ARD1), the catalytic subunit of the major N-terminal acetyltransferase complex.Semantic proximity Read ↗

dELL is an essential RNA polymerase II elongation factor with a general role in development.Semantic proximity Read ↗

Caenorhabditis elegans glp-4 Encodes a Valyl Aminoacyl tRNA Synthetase.Semantic proximity Read ↗

The Drosophila prage Gene, Required for Maternal Transcript Destabilization in Embryos, Encodes a Predicted RNA Exonuclease.Semantic proximity Read ↗

Antizyme is a target of sex-lethal in the Drosophila germline and appears to act downstream of hedgehog to regulate sex-lethal and cyclin B.Semantic proximity Read ↗

A genetic screen for maternal-effect suppressors of decapentaplegic identifies the eukaryotic translation initiation factor 4A in Drosophila.Semantic proximity Read ↗

The Ter mutation in the dead end gene causes germ cell loss and testicular germ cell tumours.Semantic proximity Read ↗

Drosophila growth and development in the absence of dMyc and dMnt.Semantic proximity Read ↗

Genetic analyses of essential genes in cytological region 61D1-2 to 61F1-2 of Drosophila melanogaster.Semantic proximity Read ↗

A Strategy To Isolate Modifiers of Caenorhabditis elegans Lethal Mutations: Investigating the Endoderm Specifying Ability of the Intestinal Differentiation GATA Factor ELT-2.Semantic proximity Read ↗

Deficiency in a glutamine-specific methyltransferase for release factor causes mouse embryonic lethality.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.