Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Exit from host cells by the pathogenic parasite Toxoplasma gondii does not require motility.

Eukaryotic cell - 1 Jan 2008

Lavine Mark D, Arrizabalaga Gustavo

Abstract excerpt

The process by which the intracellular parasite Toxoplasma gondii exits its host cell is central to its propagation and pathogenesis. Experimental induction of motility in intracellular parasites results in parasite egress, leading to the hypothesis that egress depends on the parasite's actin-dep...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Cytochalasin D
Share to Host-Parasite Interactions
Share to Ionophores

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Marine Toxins

Share to Nucleic Acid Synthesis Inhibitors

Share to Oxazoles

Share to Potassium

Share to Toxoplasma

Source

Open in PubMed Open full text

Identifiers

PMID: 17993573
DOI: 10.1128/EC.00301-07
PMCID: PMC2224157

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-17993573-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Exit from host cells by the pathogenic parasite Toxoplasma gondii does not require motility.PMID 17993573Read ↗

Toxoplasma gondii: induction of egress by the potassium ionophore nigericin.Semantic proximity Read ↗

Toxoplasma gondii Toxolysin 4 Contributes to Efficient Parasite Egress from Host CellsSemantic proximity Read ↗

Surface attachment, promoted by the actomyosin system of Toxoplasma gondii is important for efficient gliding motility and invasion.Semantic proximity Read ↗

The toxoplasma-host cell junction is anchored to the cell cortex to sustain parasite invasive forceSemantic proximity Read ↗

Ionophore-resistant mutants of Toxoplasma gondii reveal host cell permeabilization as an early event in egress.Semantic proximity Read ↗

A Toxoplasma gondii mutant defective in responding to calcium fluxes shows reduced in vivo pathogenicity.Semantic proximity Read ↗

A genetic screen to isolate Toxoplasma gondii host-cell egress mutants.Semantic proximity Read ↗

<i>Toxoplasma gondii</i> excretion of glycolytic products is associated with acidification of the parasitophorous vacuole during parasite egressSemantic proximity Read ↗

Toxofilin, a Novel Actin-binding Protein fromToxoplasma gondii, Sequesters Actin Monomers and Caps Actin FilamentsSemantic proximity Read ↗

The toxoplasma Acto-MyoA motor complex is important but not essential for gliding motility and host cell invasion.Semantic proximity Read ↗

MyosinA is a druggable target in the widespread protozoan parasite Toxoplasma gondii.Semantic proximity Read ↗

Ethanol and acetaldehyde elevate intracellular [Ca2+] and stimulate microneme discharge in Toxoplasma gondiiSemantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.