Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

EEL-1, a Hect E3 ubiquitin ligase, controls asymmetry and persistence of the SKN-1 transcription factor in the early C. elegans embryo.

Development (Cambridge, England) - 1 Jun 2007

Page Barbara D, Diede Scott J, Tenlen Jennifer R, Ferguson Edwin L

Abstract excerpt

During early divisions of the C. elegans embryo, many maternally supplied determinants accumulate asymmetrically, and this asymmetry is crucial for proper cell fate specification. SKN-1, a transcription factor whose message is maternally supplied to the embryo, specifies the mesendodermal cell fa...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Caenorhabditis elegans
Share to Caenorhabditis elegans Proteins
Share to Chromosomes

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Embryo, Nonmammalian

Share to Green Fluorescent Proteins

Share to RNA Interference

Share to Two-Hybrid System Techniques

Share to Ubiquitin-Protein Ligases

Source

Identifiers

PMID: 17537795
DOI: 10.1242/dev.02855

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 2 Sept 2026. Prepared record publication-17537795-2026-09-02.2. Manifest publication-graph-cohort-2026-09-02.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 2 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

EEL-1, a Hect E3 ubiquitin ligase, controls asymmetry and persistence of the SKN-1 transcription factor in the early C. elegans embryo.PMID 17537795Read ↗

Knockdown of SKN-1 and the Wnt effector TCF/POP-1 reveals differences in endomesoderm specification in C. briggsae as compared with C. elegans.Semantic proximity Read ↗

EFL-3/E2F7 modulates Wnt signalling by repressing the Nemo-like kinase LIT-1 during asymmetric epidermal cell division in Caenorhabditis elegans.Semantic proximity Read ↗

The Caenorhabditis elegans Skp1-related gene family: diverse functions in cell proliferation, morphogenesis, and meiosis.Semantic proximity Read ↗

The Caenorhabditis elegans ekl (enhancer of ksr-1 lethality) genes include putative components of a germline small RNA pathway.Semantic proximity Read ↗

E1 ubiquitin-activating enzyme UBA-1 plays multiple roles throughout C. elegans development.Semantic proximity Read ↗

The Ski2-family helicase Obelus regulates Crumbs alternative splicing and cell polarity.Semantic proximity Read ↗

The Bro1-domain protein, EGO-2, promotes Notch signaling in Caenorhabditis elegans.Semantic proximity Read ↗

Combinatorial decoding of the invariant C. elegans embryonic lineage in space and time.Semantic proximity Read ↗

Maternal deployment of the embryonic SKN-1-->MED-1,2 cell specification pathway in C. elegans.Semantic proximity Read ↗

eor-1 and eor-2 are required for cell-specific apoptotic death in C. elegans.Semantic proximity Read ↗

BLMP-1/Blimp-1 Regulates the Spatiotemporal Cell Migration Pattern in C. elegansSemantic proximity Read ↗

The zinc finger protein DIE-1 is required for late events during epithelial cell rearrangement in C. elegans.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.