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Article

Genetic variation in N-acetyltransferase 1 (NAT1) and 2 (NAT2) and risk of non-Hodgkin lymphoma.

Pharmacogenetics and genomics - 1 Aug 2006

Morton Lindsay M, Schenk Maryjean, Hein David W, Davis Scott, Zahm Shelia Hoar, Cozen Wendy, Cerhan James R, Hartge Patricia, Welch Robert, Chanock Stephen J, Rothman Nathaniel, Wang Sophia S

Abstract excerpt

Animal studies suggest that lymphomagenesis can be induced by exposure to carcinogenic aromatic and heterocyclic amines found in diet, cigarette smoke and the environment, but human epidemiologic investigations of these exogenous exposures have yielded conflicting results. As part of our evaluation of the role of aromatic and heterocyclic amines, which are metabolized by N-acetyltransferase (NAT) enzymes, in the...

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Source

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Identifiers

PMID: 16847422
DOI: 10.1097/01.fpc.0000215071.59836.29
PMCID: PMC1986787

Genes

NAT2
NAT1

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

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Genetic variation in N-acetyltransferase 1 (NAT1) and 2 (NAT2) and risk of non-Hodgkin lymphoma.PMID 16847422Read ↗

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