Smoking, variation in N-acetyltransferase 1 (NAT1) and 2 (NAT2), and risk of non-Hodgkin lymphoma: a pooled analysis within the InterLymph consortium.

doi:10.1007/s10552-012-0098-4

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Article

Smoking, variation in N-acetyltransferase 1 (NAT1) and 2 (NAT2), and risk of non-Hodgkin lymphoma: a pooled analysis within the InterLymph consortium.

Cancer causes & control : CCC - 1 Jan 2013

Gibson Todd M, Smedby Karin E, Skibola Christine F, Hein David W, Slager Susan L, de Sanjosé Silvia, Vajdic Claire M, Zhang Yawei, Chiu Brian C-H, Wang Sophia S, Hjalgrim Henrik, Nieters Alexandra, Bracci Paige M, Kricker Anne, Zheng Tongzhang, Kolar Carol, Cerhan James R, Darabi Hatef, Becker Nikolaus, Conde Lucia, Holford Theodore R, Weisenburger Dennis D, De Roos Anneclaire J, Butterbach Katja, Riby Jacques, Cozen Wendy, Benavente Yolanda, Palmers Casey, Holly Elizabeth A, Sampson Joshua N, Rothman Nathaniel, Armstrong Bruce K, Morton Lindsay M

Abstract excerpt

PURPOSE: Studies of smoking and risk of non-Hodgkin lymphoma (NHL) have yielded inconsistent results, possibly due to subtype heterogeneity and/or genetic variation impacting the metabolism of tobacco-derived carcinogens, including substrates of the N-acetyltransferase enzymes NAT1 and NAT2. METH...

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Identifiers

PMID: 23160945
DOI: 10.1007/s10552-012-0098-4
PMCID: PMC3529854

Genes

NAT1
NAT2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 4 Sept 2026. Prepared record publication-23160945-2026-09-04.2. Manifest publication-graph-cohort-2026-09-04.2.

Source receipt: PubMed via Helena Literature Corpus, retrieved 4 Sept 2026.

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