Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

677TT polymorphism of methylenetetrahydrofolate reductase in combination with low serum vitamin B12 is associated with coronary in-stent restenosis.

Catheterization and cardiovascular interventions : official journal of the Society for Cardiac Angiography & Interventions - 1 Mar 2006

Chung Sheng-Liang, Chiou Kuan-Rau, Charng Min-Ji

Abstract excerpt

BACKGROUND: Recent studies have shown that a common mutation (nucleotide 677 C-->T) in the methylenetetrahydrofolate reductase (MTHFR) gene may contribute to a mild rise in plasma homocysteine levels and increase the incidence of coronary artery disease. Therefore, this study was designed to further investigate whether the effects of MTHFR 677 C to T mutation, plasma homocysteine, serum vitamin B12, and folate...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Coronary Angiography
Share to Coronary Restenosis

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Folic Acid

Share to Hyperhomocysteinemia

Share to Methylenetetrahydrofolate Reductase (NADPH2)

Share to Stents

Share to Taiwan

Share to Vitamin B 12

Source

Identifiers

PMID: 16489563
DOI: 10.1002/ccd.20663

Genes

MTHFR

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

677TT polymorphism of methylenetetrahydrofolate reductase in combination with low serum vitamin B12 is associated with coronary in-stent restenosis.PMID 16489563Read ↗

Homocysteine status and polymorphisms of methylenetetrahydrofolate reductase are not associated with restenosis after stenting in coronary arteries.Semantic proximity Read ↗

Relation of a common methylenetetrahydrofolate reductase mutation and plasma homocysteine with intimal hyperplasia after coronary stenting.Semantic proximity Read ↗

MTHFR polymorphism and risk of periprocedural myocardial infarction after coronary stenting.Semantic proximity Read ↗

Correlation between C677T MTHFR gene polymorphism, plasma homocysteine levels and the incidence of CAD.Semantic proximity Read ↗

High plasma homocysteine is associated with the risk of coronary artery disease independent of methylenetetrahydrofolate reductase 677C-->T genotypes.Semantic proximity Read ↗

Methylenetetrahydrofolate reductase (MTHFR) 677C>T and methionine synthase reductase (MTRR) 66A>G polymorphisms: association with serum homocysteine and angiographic coronary artery disease in the era of flour products fortified with folic acid.Semantic proximity Read ↗

Methylenetetrahydrofolate reductase gene and coronary artery disease.Semantic proximity Read ↗

The 677C-->T mutation in the methylenetetrahydrofolate reductase gene: associations with plasma total homocysteine levels and risk of coronary atherosclerotic disease.Semantic proximity Read ↗

Thermolabile methylenetetrahydrofolate reductase in coronary artery disease.Semantic proximity Read ↗

Role of methylenetetrahydrofolate reductase 677C->T polymorphism in the development of premature myocardial infarction.Semantic proximity Read ↗

The role of hyperhomocysteinemia and methylenetetrahydrofolate reductase (MTHFR) C677T mutation in patients with retinal artery occlusion.Semantic proximity Read ↗

C677T mutation of methylenetetrahydrofolate reductase gene and serum homocysteine levels in Turkish patients with coronary artery disease.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.