Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Association study of polymorphisms in the GluR7, KA1 and KA2 kainate receptor genes (GRIK3, GRIK4, GRIK5) with schizophrenia.

Psychiatry research - 30 Jan 2006

Shibata Hiroki, Aramaki Toshihiro, Sakai Mayumi, Ninomiya Hideaki, Tashiro Nobutada, Iwata Nakao, Ozaki Norio, Fukumaki Yasuyuki

Abstract excerpt

On the basis of the glutamatergic dysfunction hypothesis of schizophrenia, we have been conducting a systematic study of the association of glutamate receptor genes with schizophrenia. Here we report association studies of schizophrenia with polymorphisms in three kainate receptor genes: GRIK3, GRIK4 and GRIK5. We selected 16, 24 and 5 common single nucleotide polymorphisms (SNPs) distributed in the entire gene...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to DNA Primers
Share to Exons

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Gene Expression

Share to Linkage Disequilibrium

Share to Receptors, Glutamate

Share to Receptors, Kainic Acid

Share to Schizophrenia

Share to Sequence Analysis, Protein

Source

Identifiers

PMID: 16325263
DOI: 10.1016/j.psychres.2005.07.015

Genes

GRIK3
GRIK4
GRIK5

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 6 Sept 2026. Manifest publication-graph-cohort-2026-09-06.1.

Source receipt: pubmed, retrieved 6 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Association study of polymorphisms in the GluR7, KA1 and KA2 kainate receptor genes (GRIK3, GRIK4, GRIK5) with schizophrenia.PMID 16325263Read ↗

Association study of polymorphisms in the GluR5 kainate receptor gene (GRIK1) with schizophrenia.Semantic proximity Read ↗

Association study of polymorphisms in the GluR6 kainate receptor gene (GRIK2) with schizophrenia.Semantic proximity Read ↗

No genetic association between polymorphisms in the kainate-type glutamate receptor gene, GRIK4, and schizophrenia in the Chinese population.Semantic proximity Read ↗

Association study of GRIK1 gene polymorphisms in schizophrenia: case-control and family-based studies.Semantic proximity Read ↗

No association between the ionotropic glutamate receptor kainate 3 gene ser310ala polymorphism and schizophrenia.Semantic proximity Read ↗

No genetic association between polymorphisms in the AMPA receptor subunit GluR4 gene (GRIA4) and schizophrenia in the Chinese population.Semantic proximity Read ↗

A possible association between schizophrenia and GRIK3 polymorphisms in a multicenter sample of Scandinavian origin (SCOPE).Semantic proximity Read ↗

Exomic sequencing of the ionotropic glutamate receptor N-methyl-D-aspartate 3A gene (GRIN3A) reveals no association with schizophrenia.Semantic proximity Read ↗

Positive association of the AMPA receptor subunit GluR4 gene (GRIA4) haplotype with schizophrenia: linkage disequilibrium mapping using SNPs evenly distributed across the gene region.Semantic proximity Read ↗

Association between the ionotropic glutamate receptor kainate3 (GRIK3) Ser310Ala polymorphism and schizophrenia in the Indian population.Semantic proximity Read ↗

No association between the metabotropic glutamate receptor type 3 gene (GRM3) and schizophrenia in a Japanese population.Semantic proximity Read ↗

Association Study of N-Methyl-D-Aspartate Receptor Subunit 2B (GRIN2B) Polymorphisms and Schizophrenia Symptoms in the Han Chinese Population.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.