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Article

Abnormal synaptic plasticity in the Ts1Cje segmental trisomy 16 mouse model of Down syndrome

29 Apr 2005

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Identifiers

PMID: 15992587
DOI: 10.1016/j.neuropharm.2005.02.012

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Abnormal synaptic plasticity in the Ts1Cje segmental trisomy 16 mouse model of Down syndromePMID 15992587Read ↗

Synaptic and cognitive abnormalities in mouse models of Down syndrome: exploring genotype-phenotype relationships.Semantic proximity Read ↗

Neocortical neuronal production and maturation defects in the TcMAC21 mouse model of Down syndromeSemantic proximity Read ↗

Abnormal expression of synaptic proteins and neurotrophin-3 in the down syndrome mouse model Ts65DnSemantic proximity Read ↗

From Abnormal Hippocampal Synaptic Plasticity in Down Syndrome Mouse Models to Cognitive Disability in Down SyndromeSemantic proximity Read ↗

Ts1Cje, a partial trisomy 16 mouse model for Down syndrome, exhibits learning and behavioral abnormalitiesSemantic proximity Read ↗

DSCAM gene triplication causes excessive GABAergic synapses in the neocortex in Down syndrome mouse modelsSemantic proximity Read ↗

Understanding mental retardation in Down's syndrome using trisomy 16 mouse modelsSemantic proximity Read ↗

The "Down syndrome critical region" is sufficient in the mouse model to confer behavioral, neurophysiological, and synaptic phenotypes characteristic of Down syndrome.Semantic proximity Read ↗

Alteration of inhibitory circuits in the somatosensory cortex of Ts65Dn mice, a model for Down’s syndromeSemantic proximity Read ↗

Impairments in motor coordination without major changes in cerebellar plasticity in the Tc1 mouse model of Down syndromeSemantic proximity Read ↗

Altered long-term potentiation in the young and old Ts65Dn mouse, a model for down syndromeSemantic proximity Read ↗

Glutamatergic synaptic deficits in the prefrontal cortex of the Ts65Dn mouse model for Down syndromeSemantic proximity Read ↗

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