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Article

TNF alpha-308 genotype and renin-angiotensin system in hemodialysis patients: an effect on inflammatory cytokine levels?

Artificial organs - 1 Feb 2005

Genctoy Gultekin, Altun Bulent, Kiykim Ahmet Alper, Arici Mustafa, Erdem Yunus, Cağlarg Meltem, Yasavul Unal, Turgan Cetin, Cağlar Sali

Abstract excerpt

BACKGROUND: Renin-angiotensin system (RAS) was suggested to modulate inflammatory cytokine production. Angiotensin II was consistently shown to increase production of tumor necrosis factor alpha (TNF-alpha). However, inflammatory cytokines and RAS were modulated by genetic polymorphisms such as TNF-alpha-308 G > A and angiotensin-converting enzyme (ACE) I/D gene polymorphisms. The aim of this study was to...

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Share to Cytokines
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Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Kidney Failure, Chronic

Share to Peptidyl-Dipeptidase A

Share to Polymorphism, Genetic

Share to Renal Dialysis

Share to Renin-Angiotensin System

Share to Tumor Necrosis Factor-alpha

Source

Identifiers

PMID: 15670287
DOI: 10.1111/j.1525-1594.2005.29029.x

Genes

TNF
ACE

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 5 Sept 2026. Manifest publication-graph-cohort-2026-09-05.1.

Source receipt: pubmed, retrieved 5 Sept 2026.

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