Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Modulation of endothelin-A receptor, Galpha subunit, and RGS2 expression during H9c2 cardiomyoblast differentiation.

Journal of receptor and signal transduction research - 1 Jan 2004

Giusti Laura, Gargini Claudia, Ceccarelli Francesca, Bacci Michela, Italiani Paola, Mazzoni Maria R

Abstract excerpt

In cardiac myocytes, growth responses depend on activation of G protein-coupled receptors interacting with Gq/11 protein subfamily members. Endothelin receptors of the ETA subtype belong to this receptor group inducing hypertrophic responses. To understand the role of ETA receptors and signal transduction proteins in modulating cell growth, we analyzed the pharmacological profile of this receptor, its level of...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Binding, Competitive
Share to Culture Media

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Fluorescent Antibody Technique, Indirect

Share to GTP-Binding Protein alpha Subunits, Gq-G11

Share to Immunoblotting

Share to Myocytes, Cardiac

Share to Receptor, Endothelin A

Share to RGS Proteins

Source

Identifiers

PMID: 15648448
DOI: 10.1081/rrs-200040331

Genes

RGS2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Modulation of endothelin-A receptor, Galpha subunit, and RGS2 expression during H9c2 cardiomyoblast differentiation.PMID 15648448Read ↗

Chronic alpha-adrenergic receptor stimulation modulates the contractile phenotype of cardiac myocytes in vitro.Semantic proximity Read ↗

Deregulation of RGS2 in cardiovascular diseasesSemantic proximity Read ↗

Various hypertrophic stimuli induce distinct phenotypes in cardiomyocytes.Semantic proximity Read ↗

Nuclear GPCRs in cardiomyocytes: an insider's view of β-adrenergic receptor signaling.Semantic proximity Read ↗

The retinoic acid response is a minor component of the cardiac phenotype in H9c2 myoblast differentiation.Semantic proximity Read ↗

Quantification of morphological, functional, and biochemical features of H9c2 rat cardiomyoblast retinoic acid differentiationSemantic proximity Read ↗

Thyroid hormone receptor alpha 1: a switch to cardiac cell "metamorphosis"?Semantic proximity Read ↗

Gene Expression Profiling of H9c2 Myoblast Differentiation towards a Cardiac-Like Phenotype.Semantic proximity Read ↗

Vitamin A as a Transcriptional Regulator of Cardiovascular DiseaseSemantic proximity Read ↗

beta-arrestin-dependent, G protein-independent ERK1/2 activation by the beta2 adrenergic receptor.Semantic proximity Read ↗

Cardiac β2-Adrenergic Receptor Phosphorylation at Ser355/356 Regulates Receptor Internalization and Functional Resensitization.Semantic proximity Read ↗

Gq signaling in cardiac adaptation and maladaptation.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.