Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Quantitative gene expression profiling implicates genes for susceptibility and resistance to alveolar bone loss.

Infection and immunity - 1 Aug 2004

Hart G T, Shaffer D J, Akilesh S, Brown A C, Moran L, Roopenian D C, Baker P J

Abstract excerpt

Periodontal disease is one of the most prevalent chronic inflammatory diseases. There is a genetic component to susceptibility and resistance to this disease. Using a mouse model, we investigated the progression of alveolar bone loss by gene expression profiling of susceptible and resistant mouse strains (BALB/cByJ and A/J, respectively). We employed a novel and sensitive quantitative real-time PCR method to...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alveolar Bone Loss
Share to Bacteroidaceae Infections

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Gingiva

Share to Mice, Inbred BALB C

Share to Mouth Diseases

Share to Oligonucleotide Array Sequence Analysis

Share to Porphyromonas gingivalis

Share to Spleen

Source

Open in PubMed Open full text

Identifiers

PMID: 15271905
DOI: 10.1128/IAI.72.8.4471-4479.2004
PMCID: PMC470695

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Quantitative gene expression profiling implicates genes for susceptibility and resistance to alveolar bone loss.PMID 15271905Read ↗

Genetic control of the immune response in pathogenesis.Semantic proximity Read ↗

Genetic control of susceptibility to Porphyromonas gingivalis-induced alveolar bone loss in mice.Semantic proximity Read ↗

Mouse gingival single-cell transcriptomic atlas: An activated fibroblast subpopulation guides oral barrier immunity in periodontitisSemantic proximity Read ↗

Fibrotic response as a distinguishing feature of resistance and susceptibility to pulmonary infection with Mycobacterium tuberculosis in mice.Semantic proximity Read ↗

Resistance and susceptibility to tuberculosis analysed at the transcriptome level: lessons from mouse macrophages.Semantic proximity Read ↗

Translation of mouse model to human gives insights into periodontitis etiology.Semantic proximity Read ↗

Differential gene expression in pristane-induced arthritis susceptible DA versus resistant E3 rats.Semantic proximity Read ↗

Gene-expression profiling of the early stages of MOG-induced EAE proves EAE-resistance as an active process.Semantic proximity Read ↗

Blood Gene Expression Signatures Predict Invasive CandidiasisSemantic proximity Read ↗

Genetic susceptibility to chronic periodontal disease.Semantic proximity Read ↗

Unique miRomics Expression Profiles in Tannerella forsythia-Infected Mandibles during Periodontitis Using Machine LearningSemantic proximity Read ↗

Host susceptibility to periodontitis: mapping murine genomic regions.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.