Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

An animal model to discern torsin function: suppression of protein aggregation in C. elegans.

Advances in neurology - 1 Jan 2004

Caldwell Guy A, Cao Songsong, Gelwix Christopher C, Sexton Elaina G, Caldwell Kim A

Abstract excerpt

No abstract is available from the source.

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Enhancer Elements, Genetic
Share to Fluorescent Antibody Technique
Share to Luminescent Proteins
Share to Models, Animal

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Phosphotransferases (Alcohol Group Acceptor)

Share to Suppression, Genetic

Share to Two-Hybrid System Techniques

Share to Ubiquitin

Source

Identifiers

PMID: 14509658

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 8 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

An animal model to discern torsin function: suppression of protein aggregation in C. elegans.PMID 14509658Read ↗

Suppression of polyglutamine-induced protein aggregation in Caenorhabditis elegans by torsin proteins.Semantic proximity Read ↗

Caenorhabditis elegans Models CountSemantic proximity Read ↗

Caenorhabditis elegans Models to Investigate the Mechanisms Underlying Tau Toxicity in TauopathiesSemantic proximity Read ↗

Genetic screens in Caenorhabditis elegans models for neurodegenerative diseasesSemantic proximity Read ↗

Hyperactive 20S Proteasome Enhances Proteostasis and ERAD in <i>C. elegans</i> via degradation of Intrinsically Disordered ProteinsSemantic proximity Read ↗

Exogenous misfolded protein oligomers can cross the intestinal barrier and cause a disease phenotype in C. elegans.Semantic proximity Read ↗

Using Caenorhabditis elegans to Screen for Tissue-Specific Chaperone Interactions.Semantic proximity Read ↗

Genetics: influence of TOR kinase on lifespan in C. elegans.Semantic proximity Read ↗

Torsin-Mediated Protection from Cellular Stress in the Dopaminergic Neurons of<i>Caenorhabditis elegans</i>Semantic proximity Read ↗

Western Blot Analysis of C. elegans Proteins.Semantic proximity Read ↗

Amyotrophic Lateral Sclerosis Mechanism: Insights from the Caenorhabditis elegans ModelsSemantic proximity Read ↗

A toolkit for GFP-mediated tissue-specific protein degradation in C. elegans.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.