Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Tumorigenicity of the met proto-oncogene and the gene for hepatocyte growth factor.

1 Nov 1992

Abstract excerpt

The met proto-oncogene is the tyrosine kinase growth factor receptor for hepatocyte growth factor/scatter factor (HGF/SF). It was previously shown that, like the oncogenic tpr-met, the mouse met proto-oncogene transforms NIH 3T3 cells. We have established NIH 3T3 cells stably expressing both human (Methu) and mouse (Metmu) met proto-oncogene products. The protein products are properly processed and appear on the...

Source

Identifiers

PMID: 1406687
DOI: 10.1128/mcb.12.11.5152

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 30 Aug 2026.

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Community interpretation

Discuss the evidence

Ask a question, add context or challenge an interpretation. Community activity is separate from scientific evidence strength.

0

discussions

0

replies

Citations encouraged

Claims remain attributable.

No discussion yet

Be the first to add a careful, cited interpretation.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Tumorigenicity of the met proto-oncogene and the gene for hepatocyte growth factor.PMID 1406687Read ↗

Tumorigenicity of the <i>met</i> Proto-Oncogene and the Gene for Hepatocyte Growth FactorSemantic proximity Read ↗

Invasiveness and metastasis of NIH 3T3 cells induced by Met-hepatocyte growth factor/scatter factor autocrine stimulation.Semantic proximity Read ↗

The roles of hepatocyte growth factor/scatter factor and met receptor in human cancers (Review).Semantic proximity Read ↗

MET: a critical player in tumorigenesis and therapeutic target.Semantic proximity Read ↗

Suppression of Ras-mediated tumorigenicity and metastasis through inhibition of the Met receptor tyrosine kinaseSemantic proximity Read ↗

Gene of the month: METSemantic proximity Read ↗

Biology of MET: a double life between normal tissue repair and tumor progression.Semantic proximity Read ↗

Activation of the JNK pathway is essential for transformation by the Met oncogene.Semantic proximity Read ↗

Evidence for a role of Met-HGF/SF during Ras-mediated tumorigenesis/metastasisSemantic proximity Read ↗

Role of the hepatocyte growth factor receptor, c-Met, in oncogenesis and potential for therapeutic inhibition.Semantic proximity Read ↗

Hepatocyte growth factor (HGF) autocrine activation predicts sensitivity to MET inhibition in glioblastomaSemantic proximity Read ↗

Autocrine hepatocyte growth factor/scatter factor-Met signaling induces transformation and the invasive/metastastic phenotype in C127 cells.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.