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Article

Interaction between Epstein-Barr virus and a T cell line (HSB-2) via a receptor phenotypically distinct from complement receptor type 2.

European journal of immunology - 1 May 1992

Hedrick J A, Watry D, Speiser C, O'Donnell P, Lambris J D, Tsoukas C D

Abstract excerpt

Epstein-Barr virus (EBV), the causative agent of mononucleosis and several human cancers, infects cells via complement receptor type 2 (CR2, CD21) which also serves as the receptor for the third complement component, C3. Expression of this receptor is restricted to B lymphocytes, immature thymocytes, and certain epithelial cells. In the present investigation; we describe the presence of a seemingly novel EBV...

Topics

Animals
Antigens, Differentiation, B-Lymphocyte
Base Sequence
Cell Line
Genes, Viral
Herpesvirus 4, Human
Molecular Sequence Data
Phenotype
Receptors, Complement
Receptors, Complement 3d

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

T-Lymphocytes

Transcription, Genetic

Source

Identifiers

PMID: 1315687
DOI: 10.1002/eji.1830220504

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 29 Aug 2026. Prepared record publication-1315687-2026-08-29.1. Manifest publication-graph-cohort-2026-08-29.1.

Source receipt: PubMed via Helena Literature Corpus, retrieved 29 Aug 2026.

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