Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

In vivo decrease in the expression of complement receptor 2 on B-cells in HIV infection.

AIDS (London, England) - 1 Jan 1993

Scott M E, Landay A L, Lint T F, Spear G T

Abstract excerpt

OBJECTIVE: To investigate changes in the expression of complement receptor 2 (CR2) on B-cells from HIV-infected individuals. CR2 is the C3d/Epstein-Barr virus receptor and has been implicated in B-cell activation. Changes in its level of expression may therefore be associated with B-cell dysfunction. DESIGN: Cross-sectional study of HIV-infected adults and age-matched control donors. METHODS: The percentage...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to B-Lymphocytes
Share to Biomarkers
Share to HIV Infections

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to HIV Seropositivity

Share to Phenotype

Source

Identifiers

PMID: 8442917
DOI: 10.1097/00002030-199301000-00006

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

In vivo decrease in the expression of complement receptor 2 on B-cells in HIV infection.PMID 8442917Read ↗

Interaction between Epstein-Barr virus and a T cell line (HSB-2) via a receptor phenotypically distinct from complement receptor type 2.Semantic proximity Read ↗

HIV-1 and HIV-2 isolates differ in their ability to activate the complement system on the surface of infected cells.Semantic proximity Read ↗

Characterization of Human Complement Receptor Type 2 (CR2/CD21) as a Receptor for IFN-α: A Potential Role in Systemic Lupus ErythematosusSemantic proximity Read ↗

Association of complement receptor 2 polymorphisms with innate resistance to HIV-1 infection.Semantic proximity Read ↗

Altered lymphocyte antigen expressions in HIV infection: a study by quantitative flow cytometry.Semantic proximity Read ↗

Complement and virus-specific antibody-dependent infection of normal B lymphocytes by human immunodeficiency virus type 1.Semantic proximity Read ↗

Structural requirements for C3d,g/Epstein-Barr virus receptor (CR2/CD21) ligand binding, internalization, and viral infection.Semantic proximity Read ↗

Isolating the Epstein-Barr Virus gp350/220 Binding Site on Complement Receptor Type 2 (CR2/CD21)Semantic proximity Read ↗

Measurement of CD69 induction in the assessment of immune function in asymptomatic HIV-infected individuals.Semantic proximity Read ↗

Molecular Basis of the Interaction between Complement Receptor Type 2 (CR2/CD21) and Epstein-Barr Virus Glycoprotein gp350Semantic proximity Read ↗

Reduction of CD55 and/or CD59 in red blood cells of patients with HIV infection.Semantic proximity Read ↗

The CR2 receptor (CD21) shows increased expression in the more differentiated cells of an antigen-specific B cell line.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.