Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

Lack of association between arylamine N-acetyltransferase 2 (NAT2) polymorphism and systemic lupus erythematosus.

Pharmacogenetics - 1 Oct 2002

Zschieschang Petra, Hiepe Falk, Gromnica-Ihle Erika, Roots Ivar, Cascorbi Ingolf

Abstract excerpt

The slow arylamine -acetyltransferase 2 (NAT2) phenotype frequently has been assumed to be associated with an elevated risk to develop a lupus-like syndrome after administration of drugs such as procainamide or hydralazine. Moreover, there are conflicting data on the role of acetylator phenotype as a susceptibility factor for systemic lupus erythematosus (SLE). Because most investigations have previously been...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Alleles
Share to Arylamine N-Acetyltransferase

Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

Share to Cohort Studies

Share to Europe

Share to Lupus Erythematosus, Systemic

Share to Odds Ratio

Share to Polymerase Chain Reaction

Share to Polymorphism, Restriction Fragment Length

Source

Identifiers

PMID: 12360107
DOI: 10.1097/00008571-200210000-00008

Genes

NAT2

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence. Graph snapshot: 2026-09-29.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

Lack of association between arylamine N-acetyltransferase 2 (NAT2) polymorphism and systemic lupus erythematosus.PMID 12360107Read ↗

Lack of association between arylamine N-acetyltransferase 2 (NAT2) polymorphism and systemic sclerosis.Semantic proximity Read ↗

Increased genotype frequency of N-acetyltransferase 2 slow acetylation in patients with rheumatoid arthritis.Semantic proximity Read ↗

Acetylation genotype and phenotype in patients with systemic lupus erythematosus.Semantic proximity Read ↗

Cigarette smoking, N-acetyltransferase 2 polymorphisms and systemic lupus erythematosus in a Japanese population.Semantic proximity Read ↗

Genotypes of polymorphic arylamine N-acetyltransferase in systemic lupus erythematosus.Semantic proximity Read ↗

Influence of polymorphic N-acetyltransferases on non-malignant spontaneous disorders and on response to drugs.Semantic proximity Read ↗

Arylamine N-acetyltransferase 2 slow acetylator polymorphisms in unrelated Iranian individuals.Semantic proximity Read ↗

Polymorphisms in NAT2 (N-acetyltransferase 2) gene in patients with systemic lupus erythematosus.Semantic proximity Read ↗

N-acetyltransferase 2 acetylation polymorphism: prevalence of slow acetylators does not differ between atopic dermatitis patients and healthy subjects.Semantic proximity Read ↗

Genotyping human polymorphic arylamine N-acetyltransferase: identification of new slow allotypic variants.Semantic proximity Read ↗

N-acetyl transferase genotypes in relation to risk of developing systemic lupus erythematosus.Semantic proximity Read ↗

Arylamine N-acetyltransferase (NAT2) gene mutations in children with allergic diseases.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.