Discover Community Create You

Loading publication...

Noodle always counts identifier-free feature events for service operation. With your permission, Google Analytics can also estimate audience and engagement. Search text, publication identifiers, cookies, IP addresses and browser fingerprints are never included in Noodle's first-party event counters.

Privacy details

Article

MYCL1, FHIT, SPARC, p16(INK4) and TP53 genes associated to lung cancer in idiopathic pulmonary fibrosis.

Journal of cellular and molecular medicine - 1 Jan 2000

Demopoulos K, Arvanitis D A, Vassilakis D A, Siafakas N M, Spandidos D A

Abstract excerpt

Idiopathic pulmonary fibrosis (IPF) is a specific form of chronic interstitial pneumonia limited to the lung and characterized by a fibroproliferative response with only minor signs of inflammation, which almost always causes rapid fibrotic destruction of the lung. In this study, we investigated genomic instability in IPF, using microsatellite DNA analysis, aiming to detect any specific genetic alterations for...

Read the complete abstract on PubMed

Topics

Share this publication in a Topic to start or enrich a Post.

Share to Acid Anhydride Hydrolases
Share to Genes, myc

Methodology Data sources Editorial policy Privacy About Contact Helena Bioinformatics

Share to Genes, p16

Share to Genes, p53

Share to Genes, Tumor Suppressor

Share to Loss of Heterozygosity

Share to Osteonectin

Share to Pulmonary Fibrosis

Source

Open in PubMed Open full text

Identifiers

PMID: 12169206
DOI: 10.1111/j.1582-4934.2002.tb00188.x
PMCID: PMC6740283

Genes

FHIT
SPARC

Provenance

PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-07.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

Related research

Semantic proximity does not establish scientific evidence.

Research trail→

Click a neighbor to travelStep 1 · 12 closest

MYCL1, FHIT, SPARC, p16(INK4) and TP53 genes associated to lung cancer in idiopathic pulmonary fibrosis.PMID 12169206Read ↗

Frequent Genetic Alterations at the Microsatellite Level in Cytologic Sputum Samples of Patients with Idiopathic Pulmonary FibrosisSemantic proximity Read ↗

Paired genetic analysis by next-generation sequencing of lung cancer and associated idiopathic pulmonary fibrosis.Semantic proximity Read ↗

Homeodomain‐interacting protein kinase2 in human idiopathic pulmonary fibrosisSemantic proximity Read ↗

Genetic variant in <i>SPDL1</i> reveals novel mechanism linking pulmonary fibrosis risk and cancer protectionSemantic proximity Read ↗

Fractional allele loss is a valuable marker for human lung cancer detection in sputum.Semantic proximity Read ↗

Global Methylation Patterns in Idiopathic Pulmonary FibrosisSemantic proximity Read ↗

Molecular genetic abnormalities in premalignant lung lesions: biological and clinical implications.Semantic proximity Read ↗

Mutation analysis of the FHIT gene in bronchoscopic specimens from patients with suspected lung cancer.Semantic proximity Read ↗

Allelic deletion analysis of the FHIT gene predicts poor survival in non-small cell lung cancer.Semantic proximity Read ↗

Lung tumours from non-smoking subjects: A p53-related genetic instability in a subset of cases.Semantic proximity Read ↗

Genetic Deletions in Sputum as Diagnostic Markers for Early Detection of Stage I Non–Small Cell Lung CancerSemantic proximity Read ↗

Connective Tissue Growth Factor Single Nucleotide Polymorphisms in (Familial) Pulmonary Fibrosis and Connective Tissue Disease Associated Interstitial Lung Disease.Semantic proximity Read ↗

⌁Select a neighboring publication to make it the new centre.