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Article

CagA status of Helicobacter pylori infection and p53 gene mutations in gastric adenocarcinoma.

Carcinogenesis - 1 Mar 2002

Shibata Atsuko, Parsonnet Julie, Longacre Teri A, Garcia Maria Isabel, Puligandla Balaram, Davis R Eric, Vogelman Joseph H, Orentreich Norman, Habel Laurel A

Abstract excerpt

Infection with Helicobacter pylori (H. pylori) increases stomach cancer risk. Helicobacter pylori strains with the cag pathogenicity island (PAI) induce more severe inflammation in the gastric epithelium and are more strongly associated with stomach cancer risk than strains lacking the PAI. We examined whether the prevalence of somatic p53 mutation in gastric adenocarcinoma differed between subjects with and...

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Methodology Data sources Editorial policy Privacy Delete account & data About Contact Helena Bioinformatics

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Identifiers

PMID: 11895856
DOI: 10.1093/carcin/23.3.419

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

Source receipt: pubmed, retrieved 7 Sept 2026.

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