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Article

In vivo electrophysiologic studies in endothelial nitric oxide synthase (eNOS)-deficient mice.

Journal of cardiovascular electrophysiology - 1 Nov 2001

Rakhit A, Maguire C T, Wakimoto H, Gehrmann J, Li G K, Kelly R A, Michel T, Berul C I

Abstract excerpt

INTRODUCTION: Endothelial nitric oxide synthase (eNOS) mediates attenuation of the L-type calcium channel and modulates myocyte contractility. Arrhythmogenic afterdepolarizations are seen in vitro in ouabain-treated isolated myocytes from eNOS-deficient mice. The aim of these studies was to characterize the baseline electrophysiologic (EP) phenotype of eNOS-deficient mice and their potential susceptibility to...

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Identifiers

PMID: 11761419
DOI: 10.1046/j.1540-8167.2001.01295.x

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PubMed metadata and Literature Corpus relationships. Corpus snapshot updated 7 Sept 2026. Manifest publication-graph-cohort-2026-09-08.1.

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